SPRR2A enhances p53 deacetylation through HDAC1 and down regulates p21 promoter activity
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2199
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
SPRR2A enhances p53 deacetylation through HDAC1 and down regulates p21 promoter activity ; volume:13 ; number:1 ; day:25 ; month:6 ; year:2012 ; pages:1-10 ; date:12.2012
BMC molecular biology ; 13, Heft 1 (25.6.2012), 1-10, 12.2012
- Creator
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Mizuguchi, Yoshiaki
Specht, Susan
Lunz, John G.
Isse, Kumiko
Corbitt, Natasha
Takizawa, Toshihiro
Demetris, Anthony J.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/1471-2199-13-20
- URN
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urn:nbn:de:101:1-2021081510514638240500
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:55 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Mizuguchi, Yoshiaki
- Specht, Susan
- Lunz, John G.
- Isse, Kumiko
- Corbitt, Natasha
- Takizawa, Toshihiro
- Demetris, Anthony J.
- SpringerLink (Online service)
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SIRT1-mediated deacetylation of FOXO3 enhances mitophagy and drives hormone resistance in endometrial cancer
Role of NAMPT, Sirt dependent deacetylation of tumor suppressors p53 and FOXO3a in AML cells
Irisin attenuates type 1 diabetic cardiomyopathy by anti-ferroptosis via SIRT1-mediated deacetylation of p53
Activation of toll like receptor 4 (TLR4) promotes cardiomyocyte apoptosis through SIRT2 dependent p53 deacetylation
Histone deacetylase 2-mediated deacetylation of the Ribonuclease 1 promoter in inflamed human endothelial cells
Mutationen des Tumorsuppressorgens p53 beim Magenkarzinom : ein Vergleich zur p53 und p21 Expression sowie zu klinisch-pathologischen Parametern
PIK3R3 inhibits cell senescence through p53/p21 signaling
JNK inhibitor IX restrains pancreatic cancer through p53 and p21
Rad9 modulates the P21 WAF1 pathway by direct association with p53
UBE2C promotes LUAD progression by ubiquitin-dependent degradation of p53 to inactivate the p53/p21 signaling pathway
MAGE-A-, p53- und p21-Expression als prognostischer Marker bei oralen Plattenepithelkarzinomen
Die Tumorsuppressorgenprodukte p53, p21 und p16 beim Mammakarzinom : Korrelationen mit klassischen Prognosefaktoren
SIRT1-mediated deacetylation of FOXO3 enhances mitophagy and drives hormone resistance in endometrial cancer
Role of NAMPT, Sirt dependent deacetylation of tumor suppressors p53 and FOXO3a in AML cells
Irisin attenuates type 1 diabetic cardiomyopathy by anti-ferroptosis via SIRT1-mediated deacetylation of p53
Activation of toll like receptor 4 (TLR4) promotes cardiomyocyte apoptosis through SIRT2 dependent p53 deacetylation
Histone deacetylase 2-mediated deacetylation of the Ribonuclease 1 promoter in inflamed human endothelial cells
Mutationen des Tumorsuppressorgens p53 beim Magenkarzinom : ein Vergleich zur p53 und p21 Expression sowie zu klinisch-pathologischen Parametern
PIK3R3 inhibits cell senescence through p53/p21 signaling
JNK inhibitor IX restrains pancreatic cancer through p53 and p21
Rad9 modulates the P21 WAF1 pathway by direct association with p53
UBE2C promotes LUAD progression by ubiquitin-dependent degradation of p53 to inactivate the p53/p21 signaling pathway
MAGE-A-, p53- und p21-Expression als prognostischer Marker bei oralen Plattenepithelkarzinomen
Die Tumorsuppressorgenprodukte p53, p21 und p16 beim Mammakarzinom : Korrelationen mit klassischen Prognosefaktoren
SIRT1-mediated deacetylation of FOXO3 enhances mitophagy and drives hormone resistance in endometrial cancer
Role of NAMPT, Sirt dependent deacetylation of tumor suppressors p53 and FOXO3a in AML cells
Irisin attenuates type 1 diabetic cardiomyopathy by anti-ferroptosis via SIRT1-mediated deacetylation of p53
Activation of toll like receptor 4 (TLR4) promotes cardiomyocyte apoptosis through SIRT2 dependent p53 deacetylation
Histone deacetylase 2-mediated deacetylation of the Ribonuclease 1 promoter in inflamed human endothelial cells