Axitinib and Sorafenib are potent in tyrosine kinase inhibitor resistant chronic myeloid leukemia cells

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Notes: Cell Communication and Signaling. 14 (2016), 6, DOI 10.1186/s12964-016-0129-y, issn: 1478-811X
IN COPYRIGHT http://rightsstatements.org/page/InC/1.0 rs

Keyword: Chronisch-myeloische Leukämie
Imatinib mesilat
Biopharmazie
Pharmakodynamik
Pharmakokinetik

Event: Veröffentlichung

(where): Freiburg

(who): Universität

(when): 2016

Creator: Halbach, Sebastian
Hu, Zehan
Gretzmeier, Christine
Ellermann, Julia
Dengjel, Jörn

Contributor: Zentrum für Biosystemanalyse
Albert-Ludwigs-Universität Freiburg. Freiburg Institute of Advanced Studies
Universitäts-Hautklinik. Freiburg im Breisgau
Albert-Ludwigs-Universität Freiburg. Institut für Molekulare Medizin und Zellforschung
Albert-Ludwigs-Universität Freiburg. Medizinische Fakultät
Albert-Ludwigs-Universität Freiburg. Fakultät für Biologie
Spemann Graduate School of Biology and Medicine
Albert-Ludwigs-Universität Freiburg

DOI: 10.1186/s12964-016-0129-y

URN: urn:nbn:de:bsz:25-freidok-125999

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 25.03.2025, 1:50 PM CET

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2016

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Axitinib and Sorafenib are potent in tyrosine kinase inhibitor resistant chronic myeloid leukemia cells

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Other Objects (12)

Gab2 and its role in Tyrosine Kinase Inhibitor Resistance in chronic myeloid leukemia

Sorafenib : Sorafenib in der Behandlung des Radiojod-refraktären differenzierten Schilddrüsenkarzinoms

Specific, targetable interactions with the microenvironment influence imatinib-resistant chronic myeloid leukemia

Comparative response of nine hepatoma cell lines to the four tyrosine kinase inhibitors, sorafenib, lenvatinib, regorafenib and cabozantinib

Analysis of the impact of Bruton Tyrosine Kinase on Myeloid Cell Development and Function

Optimierung der Sorafenib-Therapie

Discovery and mechanistic analysis of a sorafenib-derived antibiotic active against drug-resistant S. aureus, persisters and biofilms

Infliximab therapy together with tyrosine kinase inhibition targets leukemic stem cells in chronic myeloid leukemia

Investigation of resistance mechanisms in acute myeloid leukaemia towards tyrosine kinase inhibitors with mass spectrometry

Metabolic plasticity is an essential requirement of acquired tyrosine kinase inhibitor resistance in chronic myeloid leukemia

Oxidation of protein tyrosine phosphatases as an oncogenic event in FLT3-ITD positive acute myeloid leukemia

NDEL1-PDGFRB fusion gene in a myeloid malignancy with eosinophilia associated with resistance to tyrosine kinase inhibitors

Gab2 and its role in Tyrosine Kinase Inhibitor Resistance in chronic myeloid leukemia

Sorafenib : Sorafenib in der Behandlung des Radiojod-refraktären differenzierten Schilddrüsenkarzinoms

Specific, targetable interactions with the microenvironment influence imatinib-resistant chronic myeloid leukemia

Comparative response of nine hepatoma cell lines to the four tyrosine kinase inhibitors, sorafenib, lenvatinib, regorafenib and cabozantinib

Analysis of the impact of Bruton Tyrosine Kinase on Myeloid Cell Development and Function

Optimierung der Sorafenib-Therapie

Discovery and mechanistic analysis of a sorafenib-derived antibiotic active against drug-resistant S. aureus, persisters and biofilms

Infliximab therapy together with tyrosine kinase inhibition targets leukemic stem cells in chronic myeloid leukemia

Investigation of resistance mechanisms in acute myeloid leukaemia towards tyrosine kinase inhibitors with mass spectrometry

Metabolic plasticity is an essential requirement of acquired tyrosine kinase inhibitor resistance in chronic myeloid leukemia

Oxidation of protein tyrosine phosphatases as an oncogenic event in FLT3-ITD positive acute myeloid leukemia

NDEL1-PDGFRB fusion gene in a myeloid malignancy with eosinophilia associated with resistance to tyrosine kinase inhibitors

Gab2 and its role in Tyrosine Kinase Inhibitor Resistance in chronic myeloid leukemia

Sorafenib : Sorafenib in der Behandlung des Radiojod-refraktären differenzierten Schilddrüsenkarzinoms

Specific, targetable interactions with the microenvironment influence imatinib-resistant chronic myeloid leukemia

Comparative response of nine hepatoma cell lines to the four tyrosine kinase inhibitors, sorafenib, lenvatinib, regorafenib and cabozantinib

Analysis of the impact of Bruton Tyrosine Kinase on Myeloid Cell Development and Function

Optimierung der Sorafenib-Therapie

Discovery and mechanistic analysis of a sorafenib-derived antibiotic active against drug-resistant S. aureus, persisters and biofilms

Infliximab therapy together with tyrosine kinase inhibition targets leukemic stem cells in chronic myeloid leukemia

Investigation of resistance mechanisms in acute myeloid leukaemia towards tyrosine kinase inhibitors with mass spectrometry

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Oxidation of protein tyrosine phosphatases as an oncogenic event in FLT3-ITD positive acute myeloid leukemia

NDEL1-PDGFRB fusion gene in a myeloid malignancy with eosinophilia associated with resistance to tyrosine kinase inhibitors

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