Obinutuzumab-Induced B Cell Depletion Reduces Spinal Cord Pathology in a CD20 Double Transgenic Mouse Model of Multiple Sclerosis
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
-
Deutsch
- Notes
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Erlangen, Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), Dissertation, 2024
- Keyword
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Multiple Sklerose
Tiermodell
- Event
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Veröffentlichung
- (where)
-
Erlangen
- (who)
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Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU)
- (when)
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2024
- Creator
- Contributor
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Kuerten, Stefanie
Kuerten, Stefanie
Erbguth, Frank
Rothhammer, Veit
Kipp, Markus
- DOI
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10.25593/open-fau-609
- URN
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urn:nbn:de:101:1-2405100502409.454597769244
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
25.03.2025, 1:45 PM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Breakell, Thomas
- Kuerten, Stefanie
- Erbguth, Frank
- Rothhammer, Veit
- Kipp, Markus
- Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU)
Time of origin
- 2024
Other Objects (12)
Obinutuzumab-Induced B Cell Depletion Reduces Spinal Cord Pathology in a CD20 Double Transgenic Mouse Model of Multiple Sclerosis
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Neurovascular interactions in the spinal cord during embryonic development and pathology
Author correction: A deterministic genotyping workflow reduces waste of transgenic individuals by two-thirds
Amyloid pathology reduces ELP3 expression and tRNA modifications leading to impaired proteostasis
Alzheimer-like pathology in murine transgenic models: disease modification by environmental and genetic interventions
Aβ Oligomer Elimination Restores Cognition in Transgenic Alzheimer’s Mice with Full-blown Pathology
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Obinutuzumab-Induced B Cell Depletion Reduces Spinal Cord Pathology in a CD20 Double Transgenic Mouse Model of Multiple Sclerosis
Modulation of Alzheimer's pathology in transgenic mouse models
A deterministic genotyping workflow reduces waste of transgenic individuals by two-thirds
Neurovascular interactions in the spinal cord during embryonic development and pathology
Author correction: A deterministic genotyping workflow reduces waste of transgenic individuals by two-thirds
Amyloid pathology reduces ELP3 expression and tRNA modifications leading to impaired proteostasis
Alzheimer-like pathology in murine transgenic models: disease modification by environmental and genetic interventions
Aβ Oligomer Elimination Restores Cognition in Transgenic Alzheimer’s Mice with Full-blown Pathology
Molecular studies on the pathology of Alzheimer's disease with particular emphasis on transgenic mouse models
Ca2+ Depletion
Investigation of cerebellar pathology in mouse models for amyotrophic lateral sclerosis and spinal muscular atrophy
Transgenic trees
Obinutuzumab-Induced B Cell Depletion Reduces Spinal Cord Pathology in a CD20 Double Transgenic Mouse Model of Multiple Sclerosis
Modulation of Alzheimer's pathology in transgenic mouse models
A deterministic genotyping workflow reduces waste of transgenic individuals by two-thirds
Neurovascular interactions in the spinal cord during embryonic development and pathology
Author correction: A deterministic genotyping workflow reduces waste of transgenic individuals by two-thirds
Amyloid pathology reduces ELP3 expression and tRNA modifications leading to impaired proteostasis
Alzheimer-like pathology in murine transgenic models: disease modification by environmental and genetic interventions
Aβ Oligomer Elimination Restores Cognition in Transgenic Alzheimer’s Mice with Full-blown Pathology
Molecular studies on the pathology of Alzheimer's disease with particular emphasis on transgenic mouse models
Ca2+ Depletion
Investigation of cerebellar pathology in mouse models for amyotrophic lateral sclerosis and spinal muscular atrophy