MCL-1, BCL-xL and Casp-3 as key regulators in thymus homeostasis and thymic epithelial tumors
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
Göttingen, Georg-August Universität, Dissertation, 2021
- Keyword
-
Apoptosis
Krebs
Thymom
T-Lymphozyt
Zelldifferenzierung
- Event
-
Veröffentlichung
- (where)
-
Göttingen
- (who)
-
Niedersächsische Staats- und Universitätsbibliothek Göttingen
- (when)
-
2021
- Creator
- Contributor
-
Dobbelstein, Matthias
Dobbelstein, Matthias
Burfeind, Peter
Papantonis, Argyris
- URN
-
urn:nbn:de:gbv:7-21.11130/00-1735-0000-0008-59D1-8-6
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
25.03.2025, 1:41 PM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Müller, Denise
- Dobbelstein, Matthias
- Burfeind, Peter
- Papantonis, Argyris
- Niedersächsische Staats- und Universitätsbibliothek Göttingen
Time of origin
- 2021
Other Objects (12)
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Epigenetic targeting of Mcl-1 Is synthetically lethal with Bcl-xL/Bcl-2 inhibition in model systems of glioblastoma
A direct comparison of selective BH3-mimetics reveals BCL-XL, BCL-2 and MCL-1 as promising therapeutic targets in neuroblastoma
Thymic epithelial cells are a non-redundant source of Wnt ligands for thymus development
MCXLI. - MCL.
Thymic tumors
Thymic hormones
Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma
BCL-2 inhibition synergizes with dual targeting of FLT3 and AXL in FLT3 wildtype AML via downregulation of MCL-1
1221-1231, Casus MCL - MCLV
Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms
Targeting antiapoptotic proteins Bcl-xL and Mcl-1 in cholangiocarcinoma
MCL.
Epigenetic targeting of Mcl-1 Is synthetically lethal with Bcl-xL/Bcl-2 inhibition in model systems of glioblastoma
A direct comparison of selective BH3-mimetics reveals BCL-XL, BCL-2 and MCL-1 as promising therapeutic targets in neuroblastoma
Thymic epithelial cells are a non-redundant source of Wnt ligands for thymus development
MCXLI. - MCL.
Thymic tumors
Thymic hormones
Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma
BCL-2 inhibition synergizes with dual targeting of FLT3 and AXL in FLT3 wildtype AML via downregulation of MCL-1
1221-1231, Casus MCL - MCLV
Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms
Targeting antiapoptotic proteins Bcl-xL and Mcl-1 in cholangiocarcinoma
MCL.
Epigenetic targeting of Mcl-1 Is synthetically lethal with Bcl-xL/Bcl-2 inhibition in model systems of glioblastoma
A direct comparison of selective BH3-mimetics reveals BCL-XL, BCL-2 and MCL-1 as promising therapeutic targets in neuroblastoma
Thymic epithelial cells are a non-redundant source of Wnt ligands for thymus development
MCXLI. - MCL.
Thymic tumors
Thymic hormones
Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma
BCL-2 inhibition synergizes with dual targeting of FLT3 and AXL in FLT3 wildtype AML via downregulation of MCL-1