Antiangiogenic properties of axitinib versus sorafenib following sunitinib resistance in human endothelial cells — a view towards second line renal cell carcinoma treatment
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Bibliographic citation
-
In: Biomedicines, 9.2021, 11. art. 1630, S. 1-14
- Event
-
Veröffentlichung
- (where)
-
Frankfurt am Main
- (who)
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Universitätsbibliothek Johann Christian Senckenberg
- (when)
-
2021
- Creator
- DOI
-
10.3390/biomedicines9111630
- URN
-
urn:nbn:de:hebis:30:3-692732
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:32 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Jüngel, Eva
- Schnalke, Pascal
- Rutz, Jochen
- Maxeiner, Sebastian
- Chun, Felix
- Blaheta, Roman A.
- Universitätsbibliothek Johann Christian Senckenberg
Time of origin
- 2021
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Bedeutung von sE-Cadherin für die Progression des Prostatakarzinoms in vitro
Sunitinib-Sorafenib- versus Sunitinib-Everolimus-Sequenztherapie beim Nierenzellkarzinom
Sulforaphane reduces prostate cancer cell growth and proliferation in vitro by modulating the Cdk-Cyclin axis and expression of the CD44 variants 4, 5, and 7
Comparison of sunitinib and sorafenib : tumor cell response and resistance formation
Chronic sulforaphane administration inhibits resistance to the mTOR-inhibitor everolimus in bladder cancer cells
Sulforaphane impact on reactive oxygen species (ROS) in bladder carcinoma
Differential sensitivity of prostate tumor derived endothelial cells to sorafenib and sunitinib
Olive mill wastewater inhibits growth and proliferation of cisplatin- and gemcitabine-resistant bladder cancer cells in vitro by down-regulating the Akt/mTOR-signaling pathway
Tumor-endothelium cross talk blocks recruitment of neutrophils to endothelial cells: a novel mechanism of endothelial cell anergy
Growth and proliferation of renal cell carcinoma cells is blocked by low curcumin concentrations combined with visible light irradiation
Transient down-regulation of beta1 integrin subtypes on kidney carcinoma cells is induced by mechanical contact with endothelial cell membranes
Human cytomegalovirus infection alters PC3 prostate carcinoma cell adhesion to endothelial cells, extracellular matrix
Bedeutung von sE-Cadherin für die Progression des Prostatakarzinoms in vitro
Sunitinib-Sorafenib- versus Sunitinib-Everolimus-Sequenztherapie beim Nierenzellkarzinom
Sulforaphane reduces prostate cancer cell growth and proliferation in vitro by modulating the Cdk-Cyclin axis and expression of the CD44 variants 4, 5, and 7
Comparison of sunitinib and sorafenib : tumor cell response and resistance formation
Chronic sulforaphane administration inhibits resistance to the mTOR-inhibitor everolimus in bladder cancer cells
Sulforaphane impact on reactive oxygen species (ROS) in bladder carcinoma
Differential sensitivity of prostate tumor derived endothelial cells to sorafenib and sunitinib
Olive mill wastewater inhibits growth and proliferation of cisplatin- and gemcitabine-resistant bladder cancer cells in vitro by down-regulating the Akt/mTOR-signaling pathway
Tumor-endothelium cross talk blocks recruitment of neutrophils to endothelial cells: a novel mechanism of endothelial cell anergy
Growth and proliferation of renal cell carcinoma cells is blocked by low curcumin concentrations combined with visible light irradiation
Transient down-regulation of beta1 integrin subtypes on kidney carcinoma cells is induced by mechanical contact with endothelial cell membranes