Inflammatory signaling in NASH driven by hepatocyte mitochondrial dysfunctions
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
1 Online-Ressource.
- Sprache
-
Englisch
- Erschienen in
-
Inflammatory signaling in NASH driven by hepatocyte mitochondrial dysfunctions ; volume:21 ; number:1 ; day:26 ; month:10 ; year:2023 ; pages:1-16 ; date:12.2023
Journal of translational medicine ; 21, Heft 1 (26.10.2023), 1-16, 12.2023
- Urheber
-
Myint, Melissa
Oppedisano, Francesca
De Giorgi, Valeria
Kim, Byeong-Moo
Marincola, Francesco M.
Alter, Harvey J.
Nesci, Salvatore
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/s12967-023-04627-0
- URN
-
urn:nbn:de:101:1-2024011511465164527468
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:21 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Myint, Melissa
- Oppedisano, Francesca
- De Giorgi, Valeria
- Kim, Byeong-Moo
- Marincola, Francesco M.
- Alter, Harvey J.
- Nesci, Salvatore
- SpringerLink (Online service)
Ähnliche Objekte (12)
Mitochondrial Transfer by Human Mesenchymal Stromal Cells Ameliorates Hepatocyte Lipid Load in a Mouse Model of NASH
Hepatocyte phosphatase DUSP22 mitigates NASH-HCC progression by targeting FAK
Overexpression of TNFα induces senescence, autophagy and mitochondrial dysfunctions in melanoma cells
Ant1 mutant mice bridge the mitochondrial and serotonergic dysfunctions in bipolar disorder
Hepatocyte-specific IL11 cis-signaling drives lipotoxicity and underlies the transition from NAFLD to NASH
NaSH NaSH Na NASH
PM2.5 exposure increases dry eye disease risks through corneal epithelial inflammation and mitochondrial dysfunctions
Niclosamide suppresses renal cell carcinoma by inhibiting Wnt/β-catenin and inducing mitochondrial dysfunctions
GSDMD induces hepatocyte pyroptosis to trigger alcoholic hepatitis through modulating mitochondrial dysfunction
Mitochondrial IRG1 traps MCL-1 to induce hepatocyte apoptosis and promote carcinogenesis
Hepatocyte transplantation
A systematic review of nutraceutical interventions for mitochondrial dysfunctions in myalgic encephalomyelitis/chronic fatigue syndrome
Mitochondrial Transfer by Human Mesenchymal Stromal Cells Ameliorates Hepatocyte Lipid Load in a Mouse Model of NASH
Hepatocyte phosphatase DUSP22 mitigates NASH-HCC progression by targeting FAK
Overexpression of TNFα induces senescence, autophagy and mitochondrial dysfunctions in melanoma cells
Ant1 mutant mice bridge the mitochondrial and serotonergic dysfunctions in bipolar disorder
Hepatocyte-specific IL11 cis-signaling drives lipotoxicity and underlies the transition from NAFLD to NASH
NaSH NaSH Na NASH
PM2.5 exposure increases dry eye disease risks through corneal epithelial inflammation and mitochondrial dysfunctions
Niclosamide suppresses renal cell carcinoma by inhibiting Wnt/β-catenin and inducing mitochondrial dysfunctions
GSDMD induces hepatocyte pyroptosis to trigger alcoholic hepatitis through modulating mitochondrial dysfunction
Mitochondrial IRG1 traps MCL-1 to induce hepatocyte apoptosis and promote carcinogenesis
Hepatocyte transplantation
A systematic review of nutraceutical interventions for mitochondrial dysfunctions in myalgic encephalomyelitis/chronic fatigue syndrome
Mitochondrial Transfer by Human Mesenchymal Stromal Cells Ameliorates Hepatocyte Lipid Load in a Mouse Model of NASH
Hepatocyte phosphatase DUSP22 mitigates NASH-HCC progression by targeting FAK
Overexpression of TNFα induces senescence, autophagy and mitochondrial dysfunctions in melanoma cells
Ant1 mutant mice bridge the mitochondrial and serotonergic dysfunctions in bipolar disorder
Hepatocyte-specific IL11 cis-signaling drives lipotoxicity and underlies the transition from NAFLD to NASH
NaSH NaSH Na NASH
PM2.5 exposure increases dry eye disease risks through corneal epithelial inflammation and mitochondrial dysfunctions
Niclosamide suppresses renal cell carcinoma by inhibiting Wnt/β-catenin and inducing mitochondrial dysfunctions
GSDMD induces hepatocyte pyroptosis to trigger alcoholic hepatitis through modulating mitochondrial dysfunction
Mitochondrial IRG1 traps MCL-1 to induce hepatocyte apoptosis and promote carcinogenesis
Hepatocyte transplantation