Mitochondrial Transfer by Human Mesenchymal Stromal Cells Ameliorates Hepatocyte Lipid Load in a Mouse Model of NASH
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
-
In: Biomedicines : open access journal, Erscheinungsjahr: 2020, Jahrgang: 8, Heft: 9, E-ISSN: 2227-9059, Artikelnummer: 350
- Event
-
Veröffentlichung
- (where)
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Leipzig
- (who)
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Universitätsbibliothek Leipzig
- (when)
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2023
- Creator
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Hsu, Mei-Ju
Karkossa, Isabel
Schäfer, Ingo
Christ, Madlen
Kühne, Hagen
Schubert, Kristin
Rolle-Kampczyk, Ulrike E.
Kalkhof, Stefan
Nickel, Sandra
Seibel, Peter
von Bergen, Martin
Christ, Bruno
- URN
-
urn:nbn:de:bsz:15-qucosa2-846756
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:44 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Hsu, Mei-Ju
- Karkossa, Isabel
- Schäfer, Ingo
- Christ, Madlen
- Kühne, Hagen
- Schubert, Kristin
- Rolle-Kampczyk, Ulrike E.
- Kalkhof, Stefan
- Nickel, Sandra
- Seibel, Peter
- von Bergen, Martin
- Christ, Bruno
- Universitätsbibliothek Leipzig
Time of origin
- 2023
Other Objects (12)
Inflammatory signaling in NASH driven by hepatocyte mitochondrial dysfunctions
Hepatocyte phosphatase DUSP22 mitigates NASH-HCC progression by targeting FAK
Lactobacillus plantarum ameliorates NASH-related inflammation by upregulating l-arginine production
Hepatocyte-specific IL11 cis-signaling drives lipotoxicity and underlies the transition from NAFLD to NASH
Hepatocyte growth factor ameliorates dermal sclerosis in the tight-skin mouse model of scleroderma
NaSH NaSH Na NASH
Hepatocyte transplantation
Phenotype instability of hepatocyte-like cells produced by direct reprogramming of mesenchymal stromal cells
Hepatocyte-specific Sox9 knockout ameliorates acute liver injury by suppressing SHP signaling and improving mitochondrial function
Ezetimibe ameliorates atherogenic lipids profiles, insulin resistance and hepatocyte growth factor in obese patients with hypercholesterolemia
NASH
NASH
Inflammatory signaling in NASH driven by hepatocyte mitochondrial dysfunctions
Hepatocyte phosphatase DUSP22 mitigates NASH-HCC progression by targeting FAK
Lactobacillus plantarum ameliorates NASH-related inflammation by upregulating l-arginine production
Hepatocyte-specific IL11 cis-signaling drives lipotoxicity and underlies the transition from NAFLD to NASH
Hepatocyte growth factor ameliorates dermal sclerosis in the tight-skin mouse model of scleroderma
NaSH NaSH Na NASH
Hepatocyte transplantation
Phenotype instability of hepatocyte-like cells produced by direct reprogramming of mesenchymal stromal cells
Hepatocyte-specific Sox9 knockout ameliorates acute liver injury by suppressing SHP signaling and improving mitochondrial function
Ezetimibe ameliorates atherogenic lipids profiles, insulin resistance and hepatocyte growth factor in obese patients with hypercholesterolemia
NASH
NASH
Inflammatory signaling in NASH driven by hepatocyte mitochondrial dysfunctions
Hepatocyte phosphatase DUSP22 mitigates NASH-HCC progression by targeting FAK
Lactobacillus plantarum ameliorates NASH-related inflammation by upregulating l-arginine production
Hepatocyte-specific IL11 cis-signaling drives lipotoxicity and underlies the transition from NAFLD to NASH
Hepatocyte growth factor ameliorates dermal sclerosis in the tight-skin mouse model of scleroderma
NaSH NaSH Na NASH
Hepatocyte transplantation
Phenotype instability of hepatocyte-like cells produced by direct reprogramming of mesenchymal stromal cells
Hepatocyte-specific Sox9 knockout ameliorates acute liver injury by suppressing SHP signaling and improving mitochondrial function
Ezetimibe ameliorates atherogenic lipids profiles, insulin resistance and hepatocyte growth factor in obese patients with hypercholesterolemia
NASH