Hochschulschrift
Partial reprogramming of melanoma cells mimics the phenotype switch and reveals SNAI3 as a novel invasion-associated marker
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Maße
-
30 cm
- Umfang
-
IV, 123 Bl.
- Sprache
-
Englisch
- Anmerkungen
-
Ill., graph. Darst.
Heidelberg, Univ., Diss., 2015 (Nicht für den Austausch)
- Klassifikation
-
Medizin, Gesundheit
Biowissenschaften, Biologie
- Schlagwort
-
Melanoma
Reprogramming
Snai3
- Urheber
- Inhaltsverzeichnis
- Rechteinformation
-
Bei diesem Objekt liegt nur das Inhaltsverzeichnis digital vor. Der Zugriff darauf ist unbeschränkt möglich.
- Letzte Aktualisierung
-
11.06.2025, 13:49 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Objekttyp
- Hochschulschrift
Beteiligte
Ähnliche Objekte (12)
Partial reprogramming of melanoma cells mimics the phenotype switch and reveals SNAI3 as a novel invasion-associated marker
MCU controls melanoma progression through a redox‐controlled phenotype switch
Glutathione-dependent reprogramming in melanoma
BORIS/CTCFL promotes a switch from a proliferative towards an invasive phenotype in melanoma cells
Presumed Melanoma of Unknown Primary Origin Metastatic to the Choroid Mimics Primary Uveal Melanoma
Crotoxin promotes macrophage reprogramming towards an antiangiogenic phenotype
STAT3 – the switch of melanoma-associated NRAS mutations
Partial reprogramming - a model for melanoma targeted therapy resistance
Epigenetic reprogramming of melanoma cells by vitamin C treatment
ALS Mimics
ROR2, a driver of “phenotype switching” in melanoma?
Plasmodium falciparum strains spontaneously switch invasion phenotype in suspension culture
Partial reprogramming of melanoma cells mimics the phenotype switch and reveals SNAI3 as a novel invasion-associated marker
MCU controls melanoma progression through a redox‐controlled phenotype switch
Glutathione-dependent reprogramming in melanoma
BORIS/CTCFL promotes a switch from a proliferative towards an invasive phenotype in melanoma cells
Presumed Melanoma of Unknown Primary Origin Metastatic to the Choroid Mimics Primary Uveal Melanoma
Crotoxin promotes macrophage reprogramming towards an antiangiogenic phenotype
STAT3 – the switch of melanoma-associated NRAS mutations
Partial reprogramming - a model for melanoma targeted therapy resistance
Epigenetic reprogramming of melanoma cells by vitamin C treatment
ALS Mimics
ROR2, a driver of “phenotype switching” in melanoma?
Plasmodium falciparum strains spontaneously switch invasion phenotype in suspension culture
Partial reprogramming of melanoma cells mimics the phenotype switch and reveals SNAI3 as a novel invasion-associated marker
MCU controls melanoma progression through a redox‐controlled phenotype switch
Glutathione-dependent reprogramming in melanoma
BORIS/CTCFL promotes a switch from a proliferative towards an invasive phenotype in melanoma cells
Presumed Melanoma of Unknown Primary Origin Metastatic to the Choroid Mimics Primary Uveal Melanoma
Crotoxin promotes macrophage reprogramming towards an antiangiogenic phenotype
STAT3 – the switch of melanoma-associated NRAS mutations
Partial reprogramming - a model for melanoma targeted therapy resistance
Epigenetic reprogramming of melanoma cells by vitamin C treatment
ALS Mimics
ROR2, a driver of “phenotype switching” in melanoma?