Hat mitgewirkt an:
-
Monomeres C-reaktives Protein erniedrigt die Aufnahme von acetyliertem LDL in humane Endothelzellen
-
Presence of NOD2 mutations is not associated with hepatic or systemic hemodynamic abnormalities of cirrhosis
-
Influence of NOD2 risk variants on hepatic encephalopathy and association with inflammation, bacterial translocation and immune activation