Marina Jendrach
Hat mitgewirkt an:
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The mitochondrial kinase PINK1, stress response and Parkinson's disease
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Aging of different avian cultured cells : lack of ROS-induced damage and quality control mechanisms
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Both Ubiquitin Ligases FBXW8 and PARK2 Are Sequestrated into Insolubility by ATXN2 PolyQ Expansions, but Only FBXW8 Expression Is Dysregulated
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Parkinson phenotype in aged PINK1-deficient mice is accompanied by progressive mitochondrial dysfunction in absence of neurodegeneration