Hat mitgewirkt an:
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NCKAP1L defects lead to a novel syndrome combining immunodeficiency, lymphoproliferation, and hyperinflammation
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Molecular mechanisms of TNFα sensitization to fasL-induced apoptosis
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JAKs and STATs from a clinical perspective: loss-of-function mutations, gain-of-function mutations, and their multidimensional consequences
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TNFα sensitizes hepatocytes to FasL-induced apoptosis by NFκB-mediated Fas upregulation