Hat mitgewirkt an:
-
Myeloid-derived Suppressor Cells in Glioblastoma Patients - Phenotype, Function and Clinical Relevance
-
War in Ukraine: a neurosurgical perspective
-
Hyperkoagulabilität und antiödematöse Therapie im Glioblastom
-
Combined inhibition of EZH2 and CDK4/6 perturbs endoplasmic reticulum-mitochondrial homeostasis and increases antitumor activity against glioblastoma