Semaglutide modulates prothrombotic and atherosclerotic mechanisms, associated with epicardial fat, neutrophils and endothelial cells network
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Semaglutide modulates prothrombotic and atherosclerotic mechanisms, associated with epicardial fat, neutrophils and endothelial cells network ; volume:23 ; number:1 ; day:3 ; month:1 ; year:2024 ; pages:1-18 ; date:12.2024
Cardiovascular diabetology ; 23, Heft 1 (3.1.2024), 1-18, 12.2024
- Creator
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García-Vega, David
Sánchez-López, David
Rodríguez-Carnero, Gemma
Villar-Taibo, Rocío
Viñuela, Juan E.
Lestegás-Soto, Adán
Seoane-Blanco, Ana
Moure-González, María
Bravo, Susana B.
Fernández, Ángel L.
González-Juanatey, José R.
Eiras, Sonia
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12933-023-02096-9
- URN
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urn:nbn:de:101:1-2024031813333074689279
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:50 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- García-Vega, David
- Sánchez-López, David
- Rodríguez-Carnero, Gemma
- Villar-Taibo, Rocío
- Viñuela, Juan E.
- Lestegás-Soto, Adán
- Seoane-Blanco, Ana
- Moure-González, María
- Bravo, Susana B.
- Fernández, Ángel L.
- González-Juanatey, José R.
- Eiras, Sonia
- SpringerLink (Online service)
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ATP release from activated neutrophils occurs via connexin 43 and modulates adenosine-dependent endothelial cell function
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Semaglutide
Complement C5a receptor and polymorphonuclear neutrophils - accomplices in atherosclerotic lesion formation
Complement C5a receptor and polymorphonuclear neutrophils- accomplices in atherosclerotic lesion formation
Loss of thrombomodulin function modulates the stability of atherosclerotic plaques
Effect of Oral Semaglutide on Cardiovascular Parameters and Their Mechanisms in Patients with Type 2 Diabetes: Rationale and Design of the Semaglutide Anti-Atherosclerotic Mechanisms of Action Study (SAMAS)
Sodium bituminosulfonate used to treat rosacea modulates generation of inflammatory mediators by primary human neutrophils
The Pseudomonas aeruginosa lectin LecB modulates intracellular reactive oxygen species production in human neutrophils
Oral Semaglutide: Dosage in Special Situations
ATP release from activated neutrophils occurs via connexin 43 and modulates adenosine-dependent endothelial cell function
Heterogeneity Among Neutrophils
Semaglutide in Obesity: Unmet Needs in Men
ANGPTL4 stabilizes atherosclerotic plaques and modulates the phenotypic transition of vascular smooth muscle cells through KLF4 downregulation
Semaglutide
Complement C5a receptor and polymorphonuclear neutrophils - accomplices in atherosclerotic lesion formation
Complement C5a receptor and polymorphonuclear neutrophils- accomplices in atherosclerotic lesion formation
Loss of thrombomodulin function modulates the stability of atherosclerotic plaques
Effect of Oral Semaglutide on Cardiovascular Parameters and Their Mechanisms in Patients with Type 2 Diabetes: Rationale and Design of the Semaglutide Anti-Atherosclerotic Mechanisms of Action Study (SAMAS)
Sodium bituminosulfonate used to treat rosacea modulates generation of inflammatory mediators by primary human neutrophils
The Pseudomonas aeruginosa lectin LecB modulates intracellular reactive oxygen species production in human neutrophils
Oral Semaglutide: Dosage in Special Situations
ATP release from activated neutrophils occurs via connexin 43 and modulates adenosine-dependent endothelial cell function
Heterogeneity Among Neutrophils
Semaglutide in Obesity: Unmet Needs in Men