Let-7 microRNA controls invasion-promoting lysosomal changes via the oncogenic transcription factor myeloid zinc finger-1
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2157-9024
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Let-7 microRNA controls invasion-promoting lysosomal changes via the oncogenic transcription factor myeloid zinc finger-1 ; volume:7 ; number:2 ; day:3 ; month:2 ; year:2018 ; pages:1-15 ; date:2.2018
Oncogenesis ; 7, Heft 2 (3.2.2018), 1-15, 2.2018
- Creator
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Tvingsholm, Siri Amanda
- Contributor
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Hansen, Malene Bredahl
Clemmensen, Knut Kristoffer Bundgaard
Brix, Ditte Marie
Rafn, Bo
Frankel, Lisa B.
Louhimo, Riku
Moreira, José
Hautaniemi, Sampsa
Gromova, Irina
Jäättelä, Marja
Kallunki, Tuula
SpringerLink (Online service)
- DOI
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10.1038/s41389-017-0014-6
- URN
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urn:nbn:de:1111-2018041620794
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:59 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Tvingsholm, Siri Amanda
- Hansen, Malene Bredahl
- Clemmensen, Knut Kristoffer Bundgaard
- Brix, Ditte Marie
- Rafn, Bo
- Frankel, Lisa B.
- Louhimo, Riku
- Moreira, José
- Hautaniemi, Sampsa
- Gromova, Irina
- Jäättelä, Marja
- Kallunki, Tuula
- SpringerLink (Online service)
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Oncogenic LIN28:let-7 interaction, small molecule screen and preclinical characterization
Oncogenic and tumor suppressive activities of microRNA-375 in cancer
Lysosomal activation and mTOR inhibition synergize to kill myeloid leukemia cells
Antipsychotics possess anti-glioblastoma activity by disrupting lysosomal function and inhibiting oncogenic signaling by stabilizing PTEN
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The role of oncogenic Cbl mutants in Kit signaling and myeloid transformation
Autolysosomal activation combined with lysosomal destabilization efficiently targets myeloid leukemia cells for cell death
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