FAM3B/PANDER inhibits cell death and increases prostate tumor growth by modulating the expression of Bcl-2 and Bcl-XL cell survival genes
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2407
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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FAM3B/PANDER inhibits cell death and increases prostate tumor growth by modulating the expression of Bcl-2 and Bcl-XL cell survival genes ; volume:18 ; number:1 ; day:22 ; month:1 ; year:2018 ; pages:1-15 ; date:12.2018
BMC cancer ; 18, Heft 1 (22.1.2018), 1-15, 12.2018
- Keyword
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Krebs
Onkologie
- Creator
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Maciel-Silva, Paula
- Contributor
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Caldeira, Izabela
de Assis Santos, Icaro
Carreira, Ana Claudia Oliveira
Siqueira, Flavia Ramos
Antonioli, Eliane
Goldberg, Anna Carla
Belizário, José Ernesto
Garay-Malpartida, Humberto Miguel
SpringerLink (Online service)
- DOI
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10.1186/s12885-017-3950-9
- URN
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urn:nbn:de:1111-201803214256
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:57 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Maciel-Silva, Paula
- Caldeira, Izabela
- de Assis Santos, Icaro
- Carreira, Ana Claudia Oliveira
- Siqueira, Flavia Ramos
- Antonioli, Eliane
- Goldberg, Anna Carla
- Belizário, José Ernesto
- Garay-Malpartida, Humberto Miguel
- SpringerLink (Online service)
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BCL-XL directly modulates RAS signalling to favour cancer cell stemness
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Bcl-XL but Not Bcl-2 is a potential target in medulloblastoma therapy
Regulation of Bcl-xL activity in apoptosis
The roles of Bcl-xL in modulating apoptosis during development of Xenopus laevis
Bcl-xL inhibition enhances Dinaciclib-induced cell death in soft-tissue sarcomas
Transient apoptosis inhibition by Bcl-xL overexpression during hematopoietic stem cell transplantation
Treatment of lung cancer cells with bcl-2 and, or bcl-xL antisense oligonucleotides induces apoptosis and sensitises cells to cenventional therapy
Bcl-xl as the most promising Bcl-2 family member in targeted treatment of chondrosarcoma
BH4 domain peptides derived from Bcl-2/Bcl-XL as novel tools against acute pancreatitis
BCL-XL expression is essential for human erythropoiesis and engraftment of hematopoietic stem cells
BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival
BCL-XL directly modulates RAS signalling to favour cancer cell stemness
ABT-737 reverses the acquired radioresistance of breast cancer cells by targeting Bcl-2 and Bcl-xL
Bcl-XL but Not Bcl-2 is a potential target in medulloblastoma therapy
Regulation of Bcl-xL activity in apoptosis
The roles of Bcl-xL in modulating apoptosis during development of Xenopus laevis
Bcl-xL inhibition enhances Dinaciclib-induced cell death in soft-tissue sarcomas
Transient apoptosis inhibition by Bcl-xL overexpression during hematopoietic stem cell transplantation
Treatment of lung cancer cells with bcl-2 and, or bcl-xL antisense oligonucleotides induces apoptosis and sensitises cells to cenventional therapy
Bcl-xl as the most promising Bcl-2 family member in targeted treatment of chondrosarcoma
BH4 domain peptides derived from Bcl-2/Bcl-XL as novel tools against acute pancreatitis