- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2045-2322
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Inflammatory Responses Reprogram TREGS Through Impairment of Neuropilin-1 ; volume:9 ; number:1 ; day:18 ; month:7 ; year:2019 ; pages:1-12 ; date:12.2019
Scientific reports ; 9, Heft 1 (18.7.2019), 1-12, 12.2019
- Creator
-
Chen, Tim Hung-Po
Arra, Manoj
Mbalaviele, Gabriel
Swarnkar, Gaurav
Abu-Amer, Yousef
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1038/s41598-019-46934-x
- URN
-
urn:nbn:de:101:1-2019110612290522964087
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:32 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Chen, Tim Hung-Po
- Arra, Manoj
- Mbalaviele, Gabriel
- Swarnkar, Gaurav
- Abu-Amer, Yousef
- SpringerLink (Online service)
Other Objects (12)
Tregs dysfunction aggravates postoperative cognitive impairment in aged mice
Cholesterol suppresses human iTreg differentiation and nTreg function through mitochondria-related mechanisms
Isoliquiritigenin attenuates lipopolysaccharide-induced cognitive impairment through antioxidant and anti-inflammatory activity
Characterization of inflammatory RORγt+ (Th17) and regulatory Foxp3+ (Treg) T cell populations
Characterization of inflammatory RORγt+ (Th17) and regulatory Foxp3+ (Treg) T cell populations
Ultrasound inflammatory parameters and Treg/Th17 cell profiles in established rheumatoid arthritis
CD8+ Tregs ameliorate inflammatory reactions in a murine model of allergic rhinitis
Change of Peripheral Blood Treg/Thl7 in Cognitive Impairment with Chronic Renal Failure Patients
Pathological conditions re-shape physiological Tregs into pathological Tregs
Stuttgart Trauba-Treg
T-reg transcriptomic signatures identify response to check-point inhibitors
CD27 expression on Treg cells limits immune responses against tumors
Tregs dysfunction aggravates postoperative cognitive impairment in aged mice
Cholesterol suppresses human iTreg differentiation and nTreg function through mitochondria-related mechanisms
Isoliquiritigenin attenuates lipopolysaccharide-induced cognitive impairment through antioxidant and anti-inflammatory activity
Characterization of inflammatory RORγt+ (Th17) and regulatory Foxp3+ (Treg) T cell populations
Characterization of inflammatory RORγt+ (Th17) and regulatory Foxp3+ (Treg) T cell populations
Ultrasound inflammatory parameters and Treg/Th17 cell profiles in established rheumatoid arthritis
CD8+ Tregs ameliorate inflammatory reactions in a murine model of allergic rhinitis
Change of Peripheral Blood Treg/Thl7 in Cognitive Impairment with Chronic Renal Failure Patients
Pathological conditions re-shape physiological Tregs into pathological Tregs
Stuttgart Trauba-Treg
T-reg transcriptomic signatures identify response to check-point inhibitors
CD27 expression on Treg cells limits immune responses against tumors
Tregs dysfunction aggravates postoperative cognitive impairment in aged mice
Cholesterol suppresses human iTreg differentiation and nTreg function through mitochondria-related mechanisms
Isoliquiritigenin attenuates lipopolysaccharide-induced cognitive impairment through antioxidant and anti-inflammatory activity
Characterization of inflammatory RORγt+ (Th17) and regulatory Foxp3+ (Treg) T cell populations
Characterization of inflammatory RORγt+ (Th17) and regulatory Foxp3+ (Treg) T cell populations
Ultrasound inflammatory parameters and Treg/Th17 cell profiles in established rheumatoid arthritis
CD8+ Tregs ameliorate inflammatory reactions in a murine model of allergic rhinitis
Change of Peripheral Blood Treg/Thl7 in Cognitive Impairment with Chronic Renal Failure Patients
Pathological conditions re-shape physiological Tregs into pathological Tregs
Stuttgart Trauba-Treg
T-reg transcriptomic signatures identify response to check-point inhibitors