Stimulation of DNA synthesis in rat and mouse liver by various tumor promoters
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Erschienen in
-
In: In: Carcinogenesis (1987) 8, 10, 1433-7.
- Schlagwort
-
Toxikologie
Toxikologie
Vergiftung
- Ereignis
-
Veröffentlichung
- (wo)
-
Würzburg
- (wer)
-
Universität Würzburg
- (wann)
-
2012
- Urheber
-
Büsser, M. T.
Lutz, Werner K.
- URN
-
urn:nbn:de:bvb:20-opus-60908
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
25.03.2025, 13:53 MEZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Büsser, M. T.
- Lutz, Werner K.
- Universität Würzburg
Entstanden
- 2012
Ähnliche Objekte (12)
Endogenous formaldehyde does not produce detectable DNA-protein crosslinks in rat liver
Investigation of the potential for binding of di(2-ethylhexyl)phthalate (DEHP) to rat liver DNA in vivo
The relevance of covalent binding to mouse liver DNA to the carcinogenic action of hexachlorocyclohexane isomers
Covalent binding of styrene to DNA in rat and mouse
Methylation of liver DNA of rat and mouse by N-nitrosodimethylamine formed in vivo from dimethylamine and nitrite
Covalent binding of aflatoxin B1 to liver DNA in rats pretreated with ethanol
Mechanism of the carcinogenic action of benzene: irreversible binding to rat liver DNA
Saccharin does not bind to DNA of liver or bladder in the rat [Short Communication]
Dose-response relationship for chemical carcinogenesis by genotoxic agents
Dose-response relationships in chemical carcinogenesis: from DNA adducts to tumor incidence
In vivo covalent binding of organic chemicals to DNA as a quantitative indicator in the process of chemical carcinogenesis
Structural characteristics of compounds that can be activated to chemically reactive metabolites: use for a prediction of a carcinogenic potential
Endogenous formaldehyde does not produce detectable DNA-protein crosslinks in rat liver
Investigation of the potential for binding of di(2-ethylhexyl)phthalate (DEHP) to rat liver DNA in vivo
The relevance of covalent binding to mouse liver DNA to the carcinogenic action of hexachlorocyclohexane isomers
Covalent binding of styrene to DNA in rat and mouse
Methylation of liver DNA of rat and mouse by N-nitrosodimethylamine formed in vivo from dimethylamine and nitrite
Covalent binding of aflatoxin B1 to liver DNA in rats pretreated with ethanol
Mechanism of the carcinogenic action of benzene: irreversible binding to rat liver DNA
Saccharin does not bind to DNA of liver or bladder in the rat [Short Communication]
Dose-response relationship for chemical carcinogenesis by genotoxic agents
Dose-response relationships in chemical carcinogenesis: from DNA adducts to tumor incidence
In vivo covalent binding of organic chemicals to DNA as a quantitative indicator in the process of chemical carcinogenesis
Structural characteristics of compounds that can be activated to chemically reactive metabolites: use for a prediction of a carcinogenic potential
Endogenous formaldehyde does not produce detectable DNA-protein crosslinks in rat liver
Investigation of the potential for binding of di(2-ethylhexyl)phthalate (DEHP) to rat liver DNA in vivo
The relevance of covalent binding to mouse liver DNA to the carcinogenic action of hexachlorocyclohexane isomers
Covalent binding of styrene to DNA in rat and mouse
Methylation of liver DNA of rat and mouse by N-nitrosodimethylamine formed in vivo from dimethylamine and nitrite
Covalent binding of aflatoxin B1 to liver DNA in rats pretreated with ethanol
Mechanism of the carcinogenic action of benzene: irreversible binding to rat liver DNA
Saccharin does not bind to DNA of liver or bladder in the rat [Short Communication]
Dose-response relationship for chemical carcinogenesis by genotoxic agents
Dose-response relationships in chemical carcinogenesis: from DNA adducts to tumor incidence
In vivo covalent binding of organic chemicals to DNA as a quantitative indicator in the process of chemical carcinogenesis