Hochschulschrift
The initial mechanism involved in N-myc oncogene amplification in human neuroblastoma cell lines
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Dimensions
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30 cm
- Extent
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XI, 74 Bl.
- Language
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Englisch
- Notes
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Ill.
Heidelberg, Univ., Diss., 1994
- Creator
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Shibasaki, Yoshiro
- Table of contents
- Rights
-
Bei diesem Objekt liegt nur das Inhaltsverzeichnis digital vor. Der Zugriff darauf ist unbeschränkt möglich.
- Last update
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11.06.2025, 2:19 PM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Object type
- Hochschulschrift
Associated
- Shibasaki, Yoshiro
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Molecular mechanisms involved in c-Myc induced lymphomagenesis
The MYCN oncogene as a minimal residual disease marker and therapeutic target in neuroblastoma
Role of the proto-oncogene c-Myc in the development of Chlamydia trachomatis
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Kinetics of cellular oncogene expression in mouse lymphocytes II. Regulation of c-fos and c-myc gene expression
Analysis of p53 and c-MYC, two key transcription factors involved in tumorigenesis
Genetic alterations and resectability predict outcome in patients with neuroblastoma assigned to high-risk solely by MYCN amplification
MYC
Patterns of oncogene activation in human neuroblastoma cells
DDX1 co-amplification confers collateral vulnerabilities in neuroblastoma
MYCN amplification in neuroblastoma: sequence, chromatin landscape, and genome remodeling
N-Myc-induced metabolic rewiring creates novel therapeutic vulnerabilities in neuroblastoma
Molecular mechanisms involved in c-Myc induced lymphomagenesis
The MYCN oncogene as a minimal residual disease marker and therapeutic target in neuroblastoma
Role of the proto-oncogene c-Myc in the development of Chlamydia trachomatis
Zur Regulation der zellulären Oncogene c-myc und c-fos während der Entwicklung des Kleinhirns
Kinetics of cellular oncogene expression in mouse lymphocytes II. Regulation of c-fos and c-myc gene expression
Analysis of p53 and c-MYC, two key transcription factors involved in tumorigenesis
Genetic alterations and resectability predict outcome in patients with neuroblastoma assigned to high-risk solely by MYCN amplification
MYC
Patterns of oncogene activation in human neuroblastoma cells
DDX1 co-amplification confers collateral vulnerabilities in neuroblastoma
MYCN amplification in neuroblastoma: sequence, chromatin landscape, and genome remodeling
N-Myc-induced metabolic rewiring creates novel therapeutic vulnerabilities in neuroblastoma
Molecular mechanisms involved in c-Myc induced lymphomagenesis
The MYCN oncogene as a minimal residual disease marker and therapeutic target in neuroblastoma
Role of the proto-oncogene c-Myc in the development of Chlamydia trachomatis
Zur Regulation der zellulären Oncogene c-myc und c-fos während der Entwicklung des Kleinhirns
Kinetics of cellular oncogene expression in mouse lymphocytes II. Regulation of c-fos and c-myc gene expression
Analysis of p53 and c-MYC, two key transcription factors involved in tumorigenesis
Genetic alterations and resectability predict outcome in patients with neuroblastoma assigned to high-risk solely by MYCN amplification