Intracellular albumin overload elicits endoplasmic reticulum stress and PKC-delta/p38 MAPK pathway activation to induce podocyte apoptosis
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2045-2322
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Intracellular albumin overload elicits endoplasmic reticulum stress and PKC-delta/p38 MAPK pathway activation to induce podocyte apoptosis ; volume:8 ; number:1 ; day:20 ; month:12 ; year:2018 ; pages:1-9 ; date:12.2018
Scientific reports ; 8, Heft 1 (20.12.2018), 1-9, 12.2018
- Creator
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Gonçalves, Guilherme Lopes
Costa-Pessoa, Juliana Martins
Thieme, Karina
Lins, Bruna Bezerra
Oliveira-Souza, Maria
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41598-018-36933-9
- URN
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urn:nbn:de:101:1-2019020100085337965291
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:50 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Gonçalves, Guilherme Lopes
- Costa-Pessoa, Juliana Martins
- Thieme, Karina
- Lins, Bruna Bezerra
- Oliveira-Souza, Maria
- SpringerLink (Online service)
Other Objects (12)
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C1q-TNF-related protein-3 attenuates pressure overload-induced cardiac hypertrophy by suppressing the p38/CREB pathway and p38-induced ER stress
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Podocyte specific deletion of PKM2 ameliorates LPS-induced podocyte injury through beta-catenin
Podocyte-Released Migrasomes in Urine Serve as an Indicator for Early Podocyte Injury
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Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na+/H+ exchanger isoform 1 activity
Leptin-mediated downregulation of glutathione peroxidase 4 and iron overload contributing to podocyte ferroptosis in diabetic nephropathy
C1q-TNF-related protein-3 attenuates pressure overload-induced cardiac hypertrophy by suppressing the p38/CREB pathway and p38-induced ER stress
FRET-Based Screening Identifies p38 MAPK and PKC Inhibition as Targets for Prevention of Seeded α-Synuclein Aggregation
PKC-independent PI3K signalling diminishes PKC inhibitor sensitivity in uveal melanoma
Immune cell infiltration triggers heart failure after induction of pressure overload in cardiac p38 MAPKα-deficient mice
Herstellung und Etablierung von 4-Hydroxytamoxifen aktivierbaren PKCα- [PKC alpha]-, PKCβ1- [PKC beta1] und PKCδ--Fusionsproteinen [PKC delta-Fusionsproteinen]
Konditionierung des Herzens vor Infarkt : orthotope Prä- und Postkonditionierung sowie organferne Präkonditionierung ; Rolle der p38 MAPK- und der PKC-Inhibition
ARROW OVERLOAD!
Podocyte specific deletion of PKM2 ameliorates LPS-induced podocyte injury through beta-catenin
Podocyte-Released Migrasomes in Urine Serve as an Indicator for Early Podocyte Injury
14th Biennial International Podocyte Conference
Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na+/H+ exchanger isoform 1 activity
Leptin-mediated downregulation of glutathione peroxidase 4 and iron overload contributing to podocyte ferroptosis in diabetic nephropathy
C1q-TNF-related protein-3 attenuates pressure overload-induced cardiac hypertrophy by suppressing the p38/CREB pathway and p38-induced ER stress
FRET-Based Screening Identifies p38 MAPK and PKC Inhibition as Targets for Prevention of Seeded α-Synuclein Aggregation
PKC-independent PI3K signalling diminishes PKC inhibitor sensitivity in uveal melanoma
Immune cell infiltration triggers heart failure after induction of pressure overload in cardiac p38 MAPKα-deficient mice
Herstellung und Etablierung von 4-Hydroxytamoxifen aktivierbaren PKCα- [PKC alpha]-, PKCβ1- [PKC beta1] und PKCδ--Fusionsproteinen [PKC delta-Fusionsproteinen]
Konditionierung des Herzens vor Infarkt : orthotope Prä- und Postkonditionierung sowie organferne Präkonditionierung ; Rolle der p38 MAPK- und der PKC-Inhibition
ARROW OVERLOAD!
Podocyte specific deletion of PKM2 ameliorates LPS-induced podocyte injury through beta-catenin
Podocyte-Released Migrasomes in Urine Serve as an Indicator for Early Podocyte Injury