Three perspectives on the molecular basis of hypercontractility caused by hypertrophic cardiomyopathy mutations
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1432-2013
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Three perspectives on the molecular basis of hypercontractility caused by hypertrophic cardiomyopathy mutations ; day:15 ; month:2 ; year:2019 ; pages:1-17
Pflügers Archiv ; (15.2.2019), 1-17
- Creator
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Spudich, James A.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1007/s00424-019-02259-2
- URN
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urn:nbn:de:101:1-2019032219151356733043
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:53 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Spudich, James A.
- SpringerLink (Online service)
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β-Cardiac myosin hypertrophic cardiomyopathy mutations release sequestered heads and increase enzymatic activity
Diagnosis and management of hypertrophic cardiomyopathy
Adverse events in families with hypertrophic or dilated cardiomyopathy and mutations in the MYBPC3gene
Contribution of sarcomere gene mutations to left atrial function in patients with hypertrophic cardiomyopathy
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Effect of sarcomere and mitochondria-related mutations on myocardial fibrosis in patients with hypertrophic cardiomyopathy
Genetic analysis in hypertrophic cardiomyopathy : missense mutations in the ventricular myosin regulatory light chain gene
Pediatric hypertrophic cardiomyopathy caused by a novel TNNI3 variant
Fatal neonatal hypertrophic cardiomyopathy caused by compound heterozygous truncating MYBPC3 mutation
Takotsubo cardiomyopathy mimicking an apical hypertrophic cardiomyopathy
Hypertrophic Cardiomyopathy with a Twist
Pediatric patients with RASopathy-associated hypertrophic cardiomyopathy: the multifaceted consequences of PTPN11 mutations
β-Cardiac myosin hypertrophic cardiomyopathy mutations release sequestered heads and increase enzymatic activity
Diagnosis and management of hypertrophic cardiomyopathy
Adverse events in families with hypertrophic or dilated cardiomyopathy and mutations in the MYBPC3gene
Contribution of sarcomere gene mutations to left atrial function in patients with hypertrophic cardiomyopathy
Genetic analysis in hypertrophic cardiomyopathy : missense mutations in the ventricular myosin regulatory light chain gene
Effect of sarcomere and mitochondria-related mutations on myocardial fibrosis in patients with hypertrophic cardiomyopathy