- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
-
Oxysterol binding protein (OSBP) contributes to hepatitis E virus replication ; volume:21 ; number:1 ; day:22 ; month:7 ; year:2024 ; pages:1-10 ; date:12.2024
Virology journal ; 21, Heft 1 (22.7.2024), 1-10, 12.2024
- Creator
-
Lin, Shaoli
Chang, Peixi
Tsao, Shane
Aderinwale, Abigail
Sallapalli, Bhargava Teja
He, Jia
Zhang, Yanjin
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12985-024-02438-3
- URN
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urn:nbn:de:101:1-2410072105124.895930848559
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:41 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Lin, Shaoli
- Chang, Peixi
- Tsao, Shane
- Aderinwale, Abigail
- Sallapalli, Bhargava Teja
- He, Jia
- Zhang, Yanjin
- SpringerLink (Online service)
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Mathematical Modeling of Hepatitis C Virus Replication
PPP2R5D promotes hepatitis C virus infection by binding to viral NS5B and enhancing viral RNA replication
Short hairpin-looped oligodeoxynucleotides reduce hepatitis C virus replication
Efficient inhibition of hepatitis B virus replication by hepatitis delta virus ribozymes delivered by targeting retrovirus
Escape of the hepatitis B virus from the interferon system and impact on hepatitis C virus replication
Genetic determinants of host- and virus-derived insertions for hepatitis E virus replication
Determinants of efficient Hepatitis C Virus RNA replication in cell culture
Establishing novel tools for studying Hepatitis C virus replication and pathogenesis
The role of the interferon system in hepatitis-c virus replication
RNA binding protein 24 regulates the translation and replication of hepatitis C virus
RNA binding protein 24 regulates the translation and replication of hepatitis C virus
Mathematical modeling of hepatitis C virus replication
Mathematical Modeling of Hepatitis C Virus Replication
PPP2R5D promotes hepatitis C virus infection by binding to viral NS5B and enhancing viral RNA replication
Short hairpin-looped oligodeoxynucleotides reduce hepatitis C virus replication
Efficient inhibition of hepatitis B virus replication by hepatitis delta virus ribozymes delivered by targeting retrovirus
Escape of the hepatitis B virus from the interferon system and impact on hepatitis C virus replication
Genetic determinants of host- and virus-derived insertions for hepatitis E virus replication
Determinants of efficient Hepatitis C Virus RNA replication in cell culture
Establishing novel tools for studying Hepatitis C virus replication and pathogenesis