Leupaxin Promotes Bladder Cancer Proliferation, Metastasis, and Angiogenesis Through the PI3K/AKT Pathway

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: Leupaxin Promotes Bladder Cancer Proliferation, Metastasis, and Angiogenesis Through the PI3K/AKT Pathway ; volume:47 ; number:6 ; year:2018 ; pages:2250-2260
Cellular physiology and biochemistry ; 47, Heft 6 (2018), 2250-2260

Creator: Hou, Teng
Zhou, Lijie
Wang, Longwang
Kazobinka, Gallina
Chen, Yumao
Zhang, Xiaoping
Chen, Zhaohui

DOI: 10.1159/000491536

URN: urn:nbn:de:101:1-2018080200315045773465

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:31 AM CEST

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Hou, Teng
Zhou, Lijie
Wang, Longwang
Kazobinka, Gallina
Chen, Yumao
Zhang, Xiaoping
Chen, Zhaohui

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Leupaxin Promotes Bladder Cancer Proliferation, Metastasis, and Angiogenesis Through the PI3K/AKT Pathway

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Other Objects (12)

Roxarsone induces angiogenesis via PI3K/Akt signaling

Tip60 Suppresses Cholangiocarcinoma Proliferation and Metastasis via PI3k-AKT

Scutellaria barbata D.Don (SBD) extracts suppressed tumor growth, metastasis and angiogenesis in Prostate cancer via PI3K/Akt pathway

LINC01370 suppresses hepatocellular carcinoma proliferation and metastasis by regulating the PI3K/AKT pathway

Nucleolar protein 3 promotes proliferation of bladder cancer cells through the PI3K-Akt pathway

PPARγ activation serves as therapeutic strategy against bladder cancer via inhibiting PI3K-Akt signaling pathway

Tetraspanin 1 promotes epithelial-to-mesenchymal transition and metastasis of cholangiocarcinoma via PI3K/AKT signaling

LAMC2 regulates the proliferation, invasion, and metastasis of gastric cancer via PI3K/Akt signaling pathway

IL-22 activates the PI3K-AKT pathway to promote colorectal cancer cell proliferation and metastasis

Targeting DNAJC19 overcomes tumor growth and lung metastasis in NSCLC by regulating PI3K/AKT signaling

Survivin expression promotes VEGF-induced tumor angiogenesis via PI3K/Akt enhanced β-catenin/Tcf-Lef dependent transcription

TEAD4 functions as a prognostic biomarker and triggers EMT via PI3K/AKT pathway in bladder cancer

Roxarsone induces angiogenesis via PI3K/Akt signaling

Tip60 Suppresses Cholangiocarcinoma Proliferation and Metastasis via PI3k-AKT

Scutellaria barbata D.Don (SBD) extracts suppressed tumor growth, metastasis and angiogenesis in Prostate cancer via PI3K/Akt pathway

LINC01370 suppresses hepatocellular carcinoma proliferation and metastasis by regulating the PI3K/AKT pathway

Nucleolar protein 3 promotes proliferation of bladder cancer cells through the PI3K-Akt pathway

PPARγ activation serves as therapeutic strategy against bladder cancer via inhibiting PI3K-Akt signaling pathway

Tetraspanin 1 promotes epithelial-to-mesenchymal transition and metastasis of cholangiocarcinoma via PI3K/AKT signaling

LAMC2 regulates the proliferation, invasion, and metastasis of gastric cancer via PI3K/Akt signaling pathway

IL-22 activates the PI3K-AKT pathway to promote colorectal cancer cell proliferation and metastasis

Targeting DNAJC19 overcomes tumor growth and lung metastasis in NSCLC by regulating PI3K/AKT signaling

Survivin expression promotes VEGF-induced tumor angiogenesis via PI3K/Akt enhanced β-catenin/Tcf-Lef dependent transcription

TEAD4 functions as a prognostic biomarker and triggers EMT via PI3K/AKT pathway in bladder cancer

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Tip60 Suppresses Cholangiocarcinoma Proliferation and Metastasis via PI3k-AKT

Scutellaria barbata D.Don (SBD) extracts suppressed tumor growth, metastasis and angiogenesis in Prostate cancer via PI3K/Akt pathway

LINC01370 suppresses hepatocellular carcinoma proliferation and metastasis by regulating the PI3K/AKT pathway

Nucleolar protein 3 promotes proliferation of bladder cancer cells through the PI3K-Akt pathway

PPARγ activation serves as therapeutic strategy against bladder cancer via inhibiting PI3K-Akt signaling pathway

Tetraspanin 1 promotes epithelial-to-mesenchymal transition and metastasis of cholangiocarcinoma via PI3K/AKT signaling

LAMC2 regulates the proliferation, invasion, and metastasis of gastric cancer via PI3K/Akt signaling pathway

IL-22 activates the PI3K-AKT pathway to promote colorectal cancer cell proliferation and metastasis

Targeting DNAJC19 overcomes tumor growth and lung metastasis in NSCLC by regulating PI3K/AKT signaling

Survivin expression promotes VEGF-induced tumor angiogenesis via PI3K/Akt enhanced β-catenin/Tcf-Lef dependent transcription

TEAD4 functions as a prognostic biomarker and triggers EMT via PI3K/AKT pathway in bladder cancer

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