Understanding the function of Pax5 in development of docetaxel-resistant neuroendocrine-like prostate cancers
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Understanding the function of Pax5 in development of docetaxel-resistant neuroendocrine-like prostate cancers ; volume:15 ; number:8 ; day:25 ; month:8 ; year:2024 ; pages:1-18 ; date:8.2024
Cell death & disease ; 15, Heft 8 (25.8.2024), 1-18, 8.2024
- Creator
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Bhattacharya, Sreyashi
Harris, Hannah L.
Islam, Ridwan
Bodas, Sanika
Polavaram, Navatha
Mishra, Juhi
Das, Dipanwita
Seshacharyulu, Parthasarathy
Kalluchi, Achyuth
Pal, Anirban
Kohli, Manish
Lele, Subodh M.
Muders, Michael
Batra, Surinder K.
Ghosh, Paramita M.
Datta, Kaustubh
Rowley, M. Jordan
Dutta, Samikshan
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41419-024-06916-y
- URN
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urn:nbn:de:101:1-2411121027273.697394641474
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:23 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Bhattacharya, Sreyashi
- Harris, Hannah L.
- Islam, Ridwan
- Bodas, Sanika
- Polavaram, Navatha
- Mishra, Juhi
- Das, Dipanwita
- Seshacharyulu, Parthasarathy
- Kalluchi, Achyuth
- Pal, Anirban
- Kohli, Manish
- Lele, Subodh M.
- Muders, Michael
- Batra, Surinder K.
- Ghosh, Paramita M.
- Datta, Kaustubh
- Rowley, M. Jordan
- Dutta, Samikshan
- SpringerLink (Online service)
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Shikonin reduces growth of docetaxel-resistant prostate cancer cells mainly through necroptosis
Shikonin reduces growth of docetaxel-resistant prostate cancer cells mainly through necroptosis
JMJD2A participates in cytoskeletal remodeling to regulate castration-resistant prostate cancer docetaxel resistance
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External validation of risk classification in patients with docetaxel-treated castration-resistant prostate cancer
Comparison of docetaxel pharmacokinetics between castration-resistant and hormone-sensitive metastatic prostate cancer patients
Targeting the glutamine metabolism to suppress cell proliferation in mesenchymal docetaxel-resistant prostate cancer
Multiple Docetaxel Retreatments Without Prednisone for Metastatic Castration-Resistant Prostate Cancer in the Docetaxel-Only Era: Effects on PSA Kinetics and Survival
Targeting SOX4/PCK2 signaling suppresses neuroendocrine trans-differentiation of castration-resistant prostate cancer