EPIKOL, a chromatin-focused CRISPR/Cas9-based screening platform, to identify cancer-specific epigenetic vulnerabilities

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2041-4889

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: EPIKOL, a chromatin-focused CRISPR/Cas9-based screening platform, to identify cancer-specific epigenetic vulnerabilities ; volume:13 ; number:8 ; day:16 ; month:8 ; year:2022 ; pages:1-11 ; date:8.2022
Cell death & disease ; 13, Heft 8 (16.8.2022), 1-11, 8.2022

Creator: Yedier-Bayram, Ozlem
Gokbayrak, Bengul
Kayabolen, Alisan
Aksu, Ali Cenk
Cavga, Ayse Derya
Cingöz, Ahmet
Kala, Ezgi Yagmur
Karabiyik, Goktug
Günsay, Rauf
Esin, Beril
Morova, Tunc
Uyulur, Fırat
Syed, Hamzah
Philpott, Martin
Cribbs, Adam P.
Kung, Sonia H. Y.
Lack, Nathan A.
Onder, Tamer T.
Bagci-Onder, Tugba

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41419-022-05146-4

URN: urn:nbn:de:101:1-2022110318510302428744

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:30 AM CEST

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Associated

Yedier-Bayram, Ozlem
Gokbayrak, Bengul
Kayabolen, Alisan
Aksu, Ali Cenk
Cavga, Ayse Derya
Cingöz, Ahmet
Kala, Ezgi Yagmur
Karabiyik, Goktug
Günsay, Rauf
Esin, Beril
Morova, Tunc
Uyulur, Fırat
Syed, Hamzah
Philpott, Martin
Cribbs, Adam P.
Kung, Sonia H. Y.
Lack, Nathan A.
Onder, Tamer T.
Bagci-Onder, Tugba
SpringerLink (Online service)

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EPIKOL, a chromatin-focused CRISPR/Cas9-based screening platform, to identify cancer-specific epigenetic vulnerabilities

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Other Objects (12)

Computational inference of cancer-specific vulnerabilities in clinical samples

Exploiting vulnerabilities of SWI/SNF chromatin remodelling complexes for cancer therapy

Vulnerabilities

Partial gene suppression improves identification of cancer vulnerabilities when CRISPR-Cas9 knockout is pan-lethal

Exploiting vulnerabilities of bromodomain-containing chromatin remodelling complexes BAF and PBAF using inhibitors and PROTACs

Generation of CRISPR-cas9 construct for knockout of genes encoding chromatin-associated proteins

Fiscal-financial vulnerabilities

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Identification of unique rectal cancer-specific subtypes

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Vulnerabilities

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Exploiting vulnerabilities of bromodomain-containing chromatin remodelling complexes BAF and PBAF using inhibitors and PROTACs

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Partial gene suppression improves identification of cancer vulnerabilities when CRISPR-Cas9 knockout is pan-lethal

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Generation of CRISPR-cas9 construct for knockout of genes encoding chromatin-associated proteins

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CRISPR/Cas9 offers a new tool for studying the role of chromatin architecture in disease pathogenesis

Identification of unique rectal cancer-specific subtypes

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