Molecular mechanisms underlying osteoarthritis development: Notch and NF-κB

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1478-6362

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Molecular mechanisms underlying osteoarthritis development: Notch and NF-κB ; volume:19 ; number:1 ; day:15 ; month:5 ; year:2017 ; pages:1-7 ; date:12.2017
Arthritis Research & Therapy ; 19, Heft 1 (15.5.2017), 1-7, 12.2017

Keyword: Orthopädie

Creator: Saito, Taku

Contributor: Tanaka, Sakae
SpringerLink (Online service)

DOI: 10.1186/s13075-017-1296-y

URN: urn:nbn:de:1111-201705253434

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 2025-08-15T07:20:01+0200

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Saito, Taku
Tanaka, Sakae
SpringerLink (Online service)

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CHMP5 attenuates osteoarthritis via inhibiting chondrocyte apoptosis and extracellular matrix degradation: involvement of NF-κB pathway

Moderate mechanical stress suppresses chondrocyte ferroptosis in osteoarthritis by regulating NF-κB p65/GPX4 signaling pathway

miR-181a-5p targets DDX3X to inhibit the progression of osteoarthritis via NF-ΚB signaling pathway

Top Notch cancer stem cells by paracrine NF-κB signaling in breast cancer

Excessive mechanical loading promotes osteoarthritis through the gremlin-1–NF-κB pathway

Vasoactive intestinal peptide alleviates osteoarthritis effectively via inhibiting NF-κB signaling pathway

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Fargesin ameliorates osteoarthritis via macrophage reprogramming by downregulating MAPK and NF-κB pathways

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NF-κB Aktivierung durch Wachstumsfaktortyrosinkinasen

CHMP5 attenuates osteoarthritis via inhibiting chondrocyte apoptosis and extracellular matrix degradation: involvement of NF-κB pathway

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miR-181a-5p targets DDX3X to inhibit the progression of osteoarthritis via NF-ΚB signaling pathway

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Molecular mechanisms underlying osteoarthritis development: Notch and NF-κB

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Other Objects (12)

Top Notch cancer stem cells by paracrine NF-κB signaling in breast cancer

Excessive mechanical loading promotes osteoarthritis through the gremlin-1–NF-κB pathway

Vasoactive intestinal peptide alleviates osteoarthritis effectively via inhibiting NF-κB signaling pathway

LY450139 Inhibited Ti-Particle-Induced Bone Dissolution via Suppressing Notch and NF-κB Signaling Pathways

Fargesin ameliorates osteoarthritis via macrophage reprogramming by downregulating MAPK and NF-κB pathways

NUMB attenuates posttraumatic osteoarthritis by inhibiting BTRC and inactivating the NF-κB pathway

Bergenin protects against osteoarthritis by inhibiting STAT3, NF-κB and Jun pathways and suppressing osteoclastogenesis

Alternative Nf-kb Signaling in Atherogenesis

NF-κB Aktivierung durch Wachstumsfaktortyrosinkinasen

CHMP5 attenuates osteoarthritis via inhibiting chondrocyte apoptosis and extracellular matrix degradation: involvement of NF-κB pathway

Moderate mechanical stress suppresses chondrocyte ferroptosis in osteoarthritis by regulating NF-κB p65/GPX4 signaling pathway

miR-181a-5p targets DDX3X to inhibit the progression of osteoarthritis via NF-ΚB signaling pathway

Top Notch cancer stem cells by paracrine NF-κB signaling in breast cancer

Excessive mechanical loading promotes osteoarthritis through the gremlin-1–NF-κB pathway

Vasoactive intestinal peptide alleviates osteoarthritis effectively via inhibiting NF-κB signaling pathway

LY450139 Inhibited Ti-Particle-Induced Bone Dissolution via Suppressing Notch and NF-κB Signaling Pathways

Fargesin ameliorates osteoarthritis via macrophage reprogramming by downregulating MAPK and NF-κB pathways

NUMB attenuates posttraumatic osteoarthritis by inhibiting BTRC and inactivating the NF-κB pathway

Bergenin protects against osteoarthritis by inhibiting STAT3, NF-κB and Jun pathways and suppressing osteoclastogenesis

Alternative Nf-kb Signaling in Atherogenesis

NF-κB Aktivierung durch Wachstumsfaktortyrosinkinasen

CHMP5 attenuates osteoarthritis via inhibiting chondrocyte apoptosis and extracellular matrix degradation: involvement of NF-κB pathway

Moderate mechanical stress suppresses chondrocyte ferroptosis in osteoarthritis by regulating NF-κB p65/GPX4 signaling pathway

miR-181a-5p targets DDX3X to inhibit the progression of osteoarthritis via NF-ΚB signaling pathway

Top Notch cancer stem cells by paracrine NF-κB signaling in breast cancer

Excessive mechanical loading promotes osteoarthritis through the gremlin-1–NF-κB pathway

Vasoactive intestinal peptide alleviates osteoarthritis effectively via inhibiting NF-κB signaling pathway

LY450139 Inhibited Ti-Particle-Induced Bone Dissolution via Suppressing Notch and NF-κB Signaling Pathways

Fargesin ameliorates osteoarthritis via macrophage reprogramming by downregulating MAPK and NF-κB pathways

NUMB attenuates posttraumatic osteoarthritis by inhibiting BTRC and inactivating the NF-κB pathway

Bergenin protects against osteoarthritis by inhibiting STAT3, NF-κB and Jun pathways and suppressing osteoclastogenesis

Alternative Nf-kb Signaling in Atherogenesis

NF-κB Aktivierung durch Wachstumsfaktortyrosinkinasen

CHMP5 attenuates osteoarthritis via inhibiting chondrocyte apoptosis and extracellular matrix degradation: involvement of NF-κB pathway

Moderate mechanical stress suppresses chondrocyte ferroptosis in osteoarthritis by regulating NF-κB p65/GPX4 signaling pathway

miR-181a-5p targets DDX3X to inhibit the progression of osteoarthritis via NF-ΚB signaling pathway

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