Decreased CD8+T cell response to Epstein-Barr virus infected B cells in multiple sclerosis is not due to decreased HLA class I expression on B cells or monocytes
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1471-2377
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Decreased CD8+T cell response to Epstein-Barr virus infected B cells in multiple sclerosis is not due to decreased HLA class I expression on B cells or monocytes ; volume:11 ; number:1 ; day:3 ; month:8 ; year:2011 ; pages:1-6 ; date:12.2011
BMC neurology ; 11, Heft 1 (3.8.2011), 1-6, 12.2011
- Urheber
-
Pender, Michael P.
- Beteiligte Personen und Organisationen
-
Csurhes, Peter A.
Pfluger, Casey MM
Burrows, Scott R.
SpringerLink (Online service)
- DOI
-
10.1186/1471-2377-11-95
- URN
-
urn:nbn:de:1111-2016030328831
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:31 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Pender, Michael P.
- Csurhes, Peter A.
- Pfluger, Casey MM
- Burrows, Scott R.
- SpringerLink (Online service)
Ähnliche Objekte (12)
Erratum to: Cisplatin-induced mesenchymal stromal cells-mediated mechanism contributing to decreased antitumor effect in breast cancer cells
Cisplatin-induced mesenchymal stromal cells-mediated mechanism contributing to decreased antitumor effect in breast cancer cells
Decreased plasticity of aged hematopoietic stem and progenitor cells to adapt to nutrient availability
Decreased nuclear Pten in neural stem cells contributes to deficits in neuronal maturation
Altered HDL particle in sickle cell disease: decreased cholesterol content is associated with hemolysis, whereas decreased Apolipoprotein A1 is linked to inflammation
BRAF inhibition sensitizes melanoma cells to α-amanitin via decreased RNA polymerase II assembly
Aging induces B cell defects and decreased antibody responses to influenza infection and vaccination
Loss of plakoglobin promotes decreased cell-cell contact, increased invasion, and breast cancer cell dissemination in vivo
Increased glucosylceramide production leads to decreased cell energy metabolism and lowered tumor marker expression in non-cancerous liver cells
Increased glucosylceramide production leads to decreased cell energy metabolism and lowered tumor marker expression in non-cancerous liver cells
Pentoxifylline sensitizes human cervical tumor cells to cisplatin-induced apoptosis by suppressing NF-kappa B and decreased cell senescence
Nitric oxide triggers the assembly of “type II” stress granules linked to decreased cell viability
Erratum to: Cisplatin-induced mesenchymal stromal cells-mediated mechanism contributing to decreased antitumor effect in breast cancer cells
Cisplatin-induced mesenchymal stromal cells-mediated mechanism contributing to decreased antitumor effect in breast cancer cells
Decreased plasticity of aged hematopoietic stem and progenitor cells to adapt to nutrient availability
Decreased nuclear Pten in neural stem cells contributes to deficits in neuronal maturation
Altered HDL particle in sickle cell disease: decreased cholesterol content is associated with hemolysis, whereas decreased Apolipoprotein A1 is linked to inflammation
BRAF inhibition sensitizes melanoma cells to α-amanitin via decreased RNA polymerase II assembly
Aging induces B cell defects and decreased antibody responses to influenza infection and vaccination
Loss of plakoglobin promotes decreased cell-cell contact, increased invasion, and breast cancer cell dissemination in vivo
Increased glucosylceramide production leads to decreased cell energy metabolism and lowered tumor marker expression in non-cancerous liver cells
Increased glucosylceramide production leads to decreased cell energy metabolism and lowered tumor marker expression in non-cancerous liver cells
Pentoxifylline sensitizes human cervical tumor cells to cisplatin-induced apoptosis by suppressing NF-kappa B and decreased cell senescence
Nitric oxide triggers the assembly of “type II” stress granules linked to decreased cell viability
Erratum to: Cisplatin-induced mesenchymal stromal cells-mediated mechanism contributing to decreased antitumor effect in breast cancer cells
Cisplatin-induced mesenchymal stromal cells-mediated mechanism contributing to decreased antitumor effect in breast cancer cells
Decreased plasticity of aged hematopoietic stem and progenitor cells to adapt to nutrient availability
Decreased nuclear Pten in neural stem cells contributes to deficits in neuronal maturation
Altered HDL particle in sickle cell disease: decreased cholesterol content is associated with hemolysis, whereas decreased Apolipoprotein A1 is linked to inflammation
BRAF inhibition sensitizes melanoma cells to α-amanitin via decreased RNA polymerase II assembly
Aging induces B cell defects and decreased antibody responses to influenza infection and vaccination
Loss of plakoglobin promotes decreased cell-cell contact, increased invasion, and breast cancer cell dissemination in vivo
Increased glucosylceramide production leads to decreased cell energy metabolism and lowered tumor marker expression in non-cancerous liver cells
Increased glucosylceramide production leads to decreased cell energy metabolism and lowered tumor marker expression in non-cancerous liver cells
Pentoxifylline sensitizes human cervical tumor cells to cisplatin-induced apoptosis by suppressing NF-kappa B and decreased cell senescence