Correlates of Pulmonary Function in Children with Sickle Cell Disease and Elevated Fetal Hemoglobin
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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Correlates of Pulmonary Function in Children with Sickle Cell Disease and Elevated Fetal Hemoglobin ; volume:27 ; number:1 ; year:2017 ; pages:49-54
Medical principles and practice ; 27, Heft 1 (2017), 49-54
- Creator
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Adekile, Adekunle D.
Azab, Asmaa Farag
Owayed, Abdullah
Khadadah, Mousa
- DOI
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10.1159/000485801
- URN
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urn:nbn:de:101:1-20180503131
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:55 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Adekile, Adekunle D.
- Azab, Asmaa Farag
- Owayed, Abdullah
- Khadadah, Mousa
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SAR1a promoter polymorphisms are not associated with fetal hemoglobin in patients with sickle cell disease from Cameroon
Immunological Determination of Human Fetal Hemoglobin
Identifying genetic variants and pathways associated with extreme levels of fetal hemoglobin in sickle cell disease in Tanzania
Genetic reversal of the globin switch concurrently modulates both fetal and sickle hemoglobin and reduces red cell sickling
An α-chain modification rivals the effect of fetal hemoglobin in retarding the rate of sickle cell fiber formation
High fetal hemoglobin level is associated with increased risk of cerebral vasculopathy in children with sickle cell disease in Mayotte
Zinc finger nuclease-mediated gene editing in hematopoietic stem cells results in reactivation of fetal hemoglobin in sickle cell disease
Proteomic discovery in sickle cell disease: Elevated neurogranin levels in children with sickle cell disease
Fetal hemoglobin level and nutritional status in patients with sickle cell disease
The Genetic and Clinical Significance of Fetal Hemoglobin Expression in Sickle Cell Disease
Central sensitization associated with low fetal hemoglobin levels in adults with sickle cell anemia
Gum Arabic as fetal hemoglobin inducing agent in sickle cell anemia; in vivo study
SAR1a promoter polymorphisms are not associated with fetal hemoglobin in patients with sickle cell disease from Cameroon
Immunological Determination of Human Fetal Hemoglobin
Identifying genetic variants and pathways associated with extreme levels of fetal hemoglobin in sickle cell disease in Tanzania
Genetic reversal of the globin switch concurrently modulates both fetal and sickle hemoglobin and reduces red cell sickling
An α-chain modification rivals the effect of fetal hemoglobin in retarding the rate of sickle cell fiber formation
High fetal hemoglobin level is associated with increased risk of cerebral vasculopathy in children with sickle cell disease in Mayotte
Zinc finger nuclease-mediated gene editing in hematopoietic stem cells results in reactivation of fetal hemoglobin in sickle cell disease
Proteomic discovery in sickle cell disease: Elevated neurogranin levels in children with sickle cell disease
Fetal hemoglobin level and nutritional status in patients with sickle cell disease
The Genetic and Clinical Significance of Fetal Hemoglobin Expression in Sickle Cell Disease
Central sensitization associated with low fetal hemoglobin levels in adults with sickle cell anemia
Gum Arabic as fetal hemoglobin inducing agent in sickle cell anemia; in vivo study
SAR1a promoter polymorphisms are not associated with fetal hemoglobin in patients with sickle cell disease from Cameroon
Immunological Determination of Human Fetal Hemoglobin
Identifying genetic variants and pathways associated with extreme levels of fetal hemoglobin in sickle cell disease in Tanzania
Genetic reversal of the globin switch concurrently modulates both fetal and sickle hemoglobin and reduces red cell sickling
An α-chain modification rivals the effect of fetal hemoglobin in retarding the rate of sickle cell fiber formation
High fetal hemoglobin level is associated with increased risk of cerebral vasculopathy in children with sickle cell disease in Mayotte
Zinc finger nuclease-mediated gene editing in hematopoietic stem cells results in reactivation of fetal hemoglobin in sickle cell disease