Prenylation inhibitors stimulate both estrogen receptor α transcriptional activity through AF-1 and AF-2 and estrogen receptor β transcriptional activity
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1465-542X
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Prenylation inhibitors stimulate both estrogen receptor α transcriptional activity through AF-1 and AF-2 and estrogen receptor β transcriptional activity ; volume:7 ; number:1 ; day:8 ; month:11 ; year:2004 ; pages:1-11 ; date:2.2004
Breast cancer research ; 7, Heft 1 (8.11.2004), 1-11, 2.2004
- Creator
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Cestac, Philippe
- Contributor
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Sarrabayrouse, Guillaume
Médale-Giamarchi, Claire
Rochaix, Philippe
Balaguer, Patrick
Favre, Gilles
Faye, Jean-Charles
Doisneau-Sixou, Sophie
SpringerLink (Online service)
- DOI
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10.1186/bcr956
- URN
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urn:nbn:de:1111-2016032355816
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:46 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Cestac, Philippe
- Sarrabayrouse, Guillaume
- Médale-Giamarchi, Claire
- Rochaix, Philippe
- Balaguer, Patrick
- Favre, Gilles
- Faye, Jean-Charles
- Doisneau-Sixou, Sophie
- SpringerLink (Online service)
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Estrogen receptor beta inhibits transcriptional activity of hypoxia inducible factor-1 through the downregulation of arylhydrocarbon receptor nuclear translocator
Development of an orthogonal ligand-receptor pair based on synthetic estrogen analogs and engineered estrogen receptor for transcriptional regulation
Mechanistic analysis of enhancer sequences in the estrogen receptor transcriptional program
Genome-wide transcriptional regulation of estrogen receptor targets in fallopian tube cells and the role of selective estrogen receptor modulators
Estrogen receptor coregulator binding modulator (ERX-11) enhances the activity of CDK4/6 inhibitors against estrogen receptor-positive breast cancers
Estrogen rescues heart failure through estrogen receptor Beta activation
Non-coding single nucleotide variants affecting estrogen receptor binding and activity
Chemical space, diversity and activity landscape analysis of estrogen receptor binders
Functional estrogen receptor signaling pathway activity in high-grade serous ovarian carcinoma as compared to estrogen receptor protein expression by immunohistochemistry
The utility of pathway selective estrogen receptor ligands that inhibit nuclear factor-κB transcriptional activity in models of rheumatoid arthritis
Pure estrogen receptor antagonists potentiate capecitabine activity in ESR1-mutant breast cancer
A Novel Dual Antibody Staining Assay to Measure Estrogen Receptor Transcriptional Activity
Estrogen receptor beta inhibits transcriptional activity of hypoxia inducible factor-1 through the downregulation of arylhydrocarbon receptor nuclear translocator
Development of an orthogonal ligand-receptor pair based on synthetic estrogen analogs and engineered estrogen receptor for transcriptional regulation
Mechanistic analysis of enhancer sequences in the estrogen receptor transcriptional program
Genome-wide transcriptional regulation of estrogen receptor targets in fallopian tube cells and the role of selective estrogen receptor modulators
Estrogen receptor coregulator binding modulator (ERX-11) enhances the activity of CDK4/6 inhibitors against estrogen receptor-positive breast cancers
Estrogen rescues heart failure through estrogen receptor Beta activation
Non-coding single nucleotide variants affecting estrogen receptor binding and activity
Chemical space, diversity and activity landscape analysis of estrogen receptor binders
Functional estrogen receptor signaling pathway activity in high-grade serous ovarian carcinoma as compared to estrogen receptor protein expression by immunohistochemistry
The utility of pathway selective estrogen receptor ligands that inhibit nuclear factor-κB transcriptional activity in models of rheumatoid arthritis