Lipopolysaccharide induces bacterial autophagy in epithelial keratinocytes of the gingival sulcus
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2121
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Lipopolysaccharide induces bacterial autophagy in epithelial keratinocytes of the gingival sulcus ; volume:19 ; number:1 ; day:30 ; month:8 ; year:2018 ; pages:1-13 ; date:12.2018
BMC cell biology ; 19, Heft 1 (30.8.2018), 1-13, 12.2018
- Creator
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Hagio-Izaki, Kanako
- Contributor
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Yasunaga, Madoka
Yamaguchi, Masahiro
Kajiya, Hiroshi
Morita, Hiromitsu
Yoneda, Masahiro
Hirofuji, Takao
Ohno, Jun
SpringerLink (Online service)
- DOI
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10.1186/s12860-018-0168-x
- URN
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urn:nbn:de:101:1-2018102023302430794411
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:28 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Hagio-Izaki, Kanako
- Yasunaga, Madoka
- Yamaguchi, Masahiro
- Kajiya, Hiroshi
- Morita, Hiromitsu
- Yoneda, Masahiro
- Hirofuji, Takao
- Ohno, Jun
- SpringerLink (Online service)
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Protein kinase A enhances lipopolysaccharide-induced IL-6, IL-8, and PGE2 production by human gingival fibroblasts
HSP90AA1 promotes the inflammation in human gingival fibroblasts induced by Porphyromonas gingivalis lipopolysaccharide via regulating of autophagy
Epinephrine penetrates through gingival sulcus unlike keratinized gingiva and evokes remote vasoconstriction in human
HMGB1 mediates lipopolysaccharide-induced macrophage autophagy and pyroptosis
Adherence of human oral keratinocytes and gingival fibroblasts to nano-structured titanium surfaces
Autophagy drives osteogenic differentiation of human gingival mesenchymal stem cells
Lipopolysaccharide exacerbates phenotypic changes of Brugada syndrome through autophagy
The Effects of Abutment Finish Lines on the Penetration Characteristics of Elastomers into the Simulated Gingival Sulcus
Fig. 189. Sulcus lingualis.
Magenstraße und Sulcus salivalis
Das Sulcus-ulnaris-Syndrom
Dose-dependent green tea effect on decrease of inflammation in human oral gingival epithelial keratinocytes: in vitro study
Protein kinase A enhances lipopolysaccharide-induced IL-6, IL-8, and PGE2 production by human gingival fibroblasts
HSP90AA1 promotes the inflammation in human gingival fibroblasts induced by Porphyromonas gingivalis lipopolysaccharide via regulating of autophagy
Epinephrine penetrates through gingival sulcus unlike keratinized gingiva and evokes remote vasoconstriction in human
HMGB1 mediates lipopolysaccharide-induced macrophage autophagy and pyroptosis
Adherence of human oral keratinocytes and gingival fibroblasts to nano-structured titanium surfaces
Autophagy drives osteogenic differentiation of human gingival mesenchymal stem cells
Lipopolysaccharide exacerbates phenotypic changes of Brugada syndrome through autophagy
The Effects of Abutment Finish Lines on the Penetration Characteristics of Elastomers into the Simulated Gingival Sulcus
Fig. 189. Sulcus lingualis.
Magenstraße und Sulcus salivalis
Das Sulcus-ulnaris-Syndrom
Dose-dependent green tea effect on decrease of inflammation in human oral gingival epithelial keratinocytes: in vitro study