ALAIN01—Alemtuzumab in autoimmune inflammatory neurodegeneration: mechanisms of action and neuroprotective potential

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1471-2377

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: ALAIN01—Alemtuzumab in autoimmune inflammatory neurodegeneration: mechanisms of action and neuroprotective potential ; volume:16 ; number:1 ; day:10 ; month:3 ; year:2016 ; pages:1-12 ; date:12.2016
BMC neurology ; 16, Heft 1 (10.3.2016), 1-12, 12.2016

Creator: Ruck, Tobias

Contributor: Afzali, Ali Maisam
Lukat, Karl-Friedrich
Eveslage, Maria
Gross, Catharina C.
Pfeuffer, Steffen
Bittner, Stefan
Klotz, Luisa
Melzer, Nico
Wiendl, Heinz
Meuth, Sven
SpringerLink (Online service)

DOI: 10.1186/s12883-016-0556-9

URN: urn:nbn:de:1111-2016080630448

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:57 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Ruck, Tobias
Afzali, Ali Maisam
Lukat, Karl-Friedrich
Eveslage, Maria
Gross, Catharina C.
Pfeuffer, Steffen
Bittner, Stefan
Klotz, Luisa
Melzer, Nico
Wiendl, Heinz
Meuth, Sven
SpringerLink (Online service)

Other Objects (12)

Evans syndrome associated with sterile inflammation of the central nervous system: a case report

Alemtuzumab : humanisierter monoklonaler Antikörper in der MS-Therapie

Erratum to: Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)

Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)

Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+T cell attack in the CNS gray matter

Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+ T cell attack in the CNS gray matter

CD4+ CD25+ FoxP3+ regulatory T cells suppress cytotoxicity of CD8+ effector T cells: implications for their capacity to limit inflammatory central nervous system damage at the parenchymal level

Hochschulschrift

Die Rolle des Zwei-Poren K+-Kanals TASK1 in der autoimmunen Neuroinflammation

Hochschulschrift

Wave-dynamical properties of dielectric resonators investigated with microwaves

Hochschulschrift

Wave-dynamical properties of dielectric resonators investigated with microwaves

Generating microglia from human pluripotent stem cells: novel in vitro models for the study of neurodegeneration

Murine K2P5.1 Deficiency Has No Impact on Autoimmune Neuroinflammation due to Compensatory K2P3.1-and KV1.3-Dependent Mechanisms

Evans syndrome associated with sterile inflammation of the central nervous system: a case report

Alemtuzumab : humanisierter monoklonaler Antikörper in der MS-Therapie

Erratum to: Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)

Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)

Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+T cell attack in the CNS gray matter

Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+ T cell attack in the CNS gray matter

CD4+ CD25+ FoxP3+ regulatory T cells suppress cytotoxicity of CD8+ effector T cells: implications for their capacity to limit inflammatory central nervous system damage at the parenchymal level

Hochschulschrift

Die Rolle des Zwei-Poren K+-Kanals TASK1 in der autoimmunen Neuroinflammation

Hochschulschrift

Wave-dynamical properties of dielectric resonators investigated with microwaves

Hochschulschrift

Wave-dynamical properties of dielectric resonators investigated with microwaves

Generating microglia from human pluripotent stem cells: novel in vitro models for the study of neurodegeneration

Murine K2P5.1 Deficiency Has No Impact on Autoimmune Neuroinflammation due to Compensatory K2P3.1-and KV1.3-Dependent Mechanisms

Evans syndrome associated with sterile inflammation of the central nervous system: a case report

Alemtuzumab : humanisierter monoklonaler Antikörper in der MS-Therapie

Erratum to: Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)

Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)

Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+T cell attack in the CNS gray matter

Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+ T cell attack in the CNS gray matter

CD4+ CD25+ FoxP3+ regulatory T cells suppress cytotoxicity of CD8+ effector T cells: implications for their capacity to limit inflammatory central nervous system damage at the parenchymal level

Hochschulschrift

Die Rolle des Zwei-Poren K+-Kanals TASK1 in der autoimmunen Neuroinflammation

Hochschulschrift

Wave-dynamical properties of dielectric resonators investigated with microwaves

Hochschulschrift

Wave-dynamical properties of dielectric resonators investigated with microwaves

Generating microglia from human pluripotent stem cells: novel in vitro models for the study of neurodegeneration

Murine K2P5.1 Deficiency Has No Impact on Autoimmune Neuroinflammation due to Compensatory K2P3.1-and KV1.3-Dependent Mechanisms

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

ALAIN01—Alemtuzumab in autoimmune inflammatory neurodegeneration: mechanisms of action and neuroprotective potential

Object Details

References and Relationships

Contributors, Places and Time

Further information

Data provider

Associated

Other Objects (12)

Evans syndrome associated with sterile inflammation of the central nervous system: a case report

Alemtuzumab : humanisierter monoklonaler Antikörper in der MS-Therapie

Erratum to: Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)

Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)

Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+T cell attack in the CNS gray matter

Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+ T cell attack in the CNS gray matter

CD4+ CD25+ FoxP3+ regulatory T cells suppress cytotoxicity of CD8+ effector T cells: implications for their capacity to limit inflammatory central nervous system damage at the parenchymal level

Die Rolle des Zwei-Poren K+-Kanals TASK1 in der autoimmunen Neuroinflammation

Wave-dynamical properties of dielectric resonators investigated with microwaves

Wave-dynamical properties of dielectric resonators investigated with microwaves

Generating microglia from human pluripotent stem cells: novel in vitro models for the study of neurodegeneration

Murine K2P5.1 Deficiency Has No Impact on Autoimmune Neuroinflammation due to Compensatory K2P3.1-and KV1.3-Dependent Mechanisms

Evans syndrome associated with sterile inflammation of the central nervous system: a case report

Alemtuzumab : humanisierter monoklonaler Antikörper in der MS-Therapie

Erratum to: Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)

Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)

Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+T cell attack in the CNS gray matter

Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+ T cell attack in the CNS gray matter

CD4+ CD25+ FoxP3+ regulatory T cells suppress cytotoxicity of CD8+ effector T cells: implications for their capacity to limit inflammatory central nervous system damage at the parenchymal level

Die Rolle des Zwei-Poren K+-Kanals TASK1 in der autoimmunen Neuroinflammation

Wave-dynamical properties of dielectric resonators investigated with microwaves

Wave-dynamical properties of dielectric resonators investigated with microwaves

Generating microglia from human pluripotent stem cells: novel in vitro models for the study of neurodegeneration

Murine K2P5.1 Deficiency Has No Impact on Autoimmune Neuroinflammation due to Compensatory K2P3.1-and KV1.3-Dependent Mechanisms

Evans syndrome associated with sterile inflammation of the central nervous system: a case report

Alemtuzumab : humanisierter monoklonaler Antikörper in der MS-Therapie

Erratum to: Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)

Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)

Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+T cell attack in the CNS gray matter

Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+ T cell attack in the CNS gray matter

CD4+ CD25+ FoxP3+ regulatory T cells suppress cytotoxicity of CD8+ effector T cells: implications for their capacity to limit inflammatory central nervous system damage at the parenchymal level

Die Rolle des Zwei-Poren K+-Kanals TASK1 in der autoimmunen Neuroinflammation

Wave-dynamical properties of dielectric resonators investigated with microwaves

Wave-dynamical properties of dielectric resonators investigated with microwaves

Generating microglia from human pluripotent stem cells: novel in vitro models for the study of neurodegeneration

Murine K2P5.1 Deficiency Has No Impact on Autoimmune Neuroinflammation due to Compensatory K2P3.1-and KV1.3-Dependent Mechanisms

Related objects

Reset password