Inducible nitric oxide synthase (iNOS) expression in monocytes during acute Dengue Fever in patients and during in vitro infection
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2334
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Inducible nitric oxide synthase (iNOS) expression in monocytes during acute Dengue Fever in patients and during in vitro infection ; volume:5 ; number:1 ; day:18 ; month:8 ; year:2005 ; pages:1-12 ; date:12.2005
BMC infectious diseases ; 5, Heft 1 (18.8.2005), 1-12, 12.2005
- Creator
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Neves-Souza, Patrícia CF
- Contributor
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Azeredo, Elzinandes L.
Zagne, Sonia MO
Valls-de-Souza, Rogério
Reis, Sonia RNI
Cerqueira, Denise IS
Nogueira, Rita MR
Kubelka, Claire F.
SpringerLink (Online service)
- DOI
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10.1186/1471-2334-5-64
- URN
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urn:nbn:de:1111-2016030247432
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:22 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Neves-Souza, Patrícia CF
- Azeredo, Elzinandes L.
- Zagne, Sonia MO
- Valls-de-Souza, Rogério
- Reis, Sonia RNI
- Cerqueira, Denise IS
- Nogueira, Rita MR
- Kubelka, Claire F.
- SpringerLink (Online service)
Other Objects (12)
The Role of Inducible Nitric Oxide Synthase (iNOS) in Human Myocardial Infarction
Tissue expression and cytokine regulation of the human inducible nitric oxide synthase (iNOS)
Inducible nitric oxide synthase inhibits macrophage migration, a potential explanation for iNOS's proatherosclerotic action
Inducible Nitric Oxide Synthase inhibits macrophage migration,a potential explanation for iNOS's proatherosclerotic action
Inducible Nitric Oxide Synthase (iNOS) Is a Novel Negative Regulator of Hematopoietic Stem/Progenitor Cell Trafficking
Reproducibility and intraindividual variation over days in buccal cell DNA methylation of two asthma genes, interferon γ (IFNγ) and inducible nitric oxide synthase (iNOS)
Modulation of inducible nitric oxide synthase expression by sumoylation
Monocytes differentiate along two alternative pathways during sterile inflammation
Malarial pigment haemozoin, IFN-gamma, TNF-alpha, IL-1beta and LPS do not stimulate expression of inducible nitric oxide synthase and production of nitric oxide in immuno-purified human monocytes
Type I interferon supports inducible nitric oxide synthase in murine hepatoma cells and hepatocytes and during experimental acetaminophen-induced liver damage
P2Y2 impacts inflammatory monocytes during acute Graft-versus-Host Disease
The role of nitric oxide during embryonic wound healing
The Role of Inducible Nitric Oxide Synthase (iNOS) in Human Myocardial Infarction
Tissue expression and cytokine regulation of the human inducible nitric oxide synthase (iNOS)
Inducible nitric oxide synthase inhibits macrophage migration, a potential explanation for iNOS's proatherosclerotic action
Inducible Nitric Oxide Synthase inhibits macrophage migration,a potential explanation for iNOS's proatherosclerotic action
Inducible Nitric Oxide Synthase (iNOS) Is a Novel Negative Regulator of Hematopoietic Stem/Progenitor Cell Trafficking
Reproducibility and intraindividual variation over days in buccal cell DNA methylation of two asthma genes, interferon γ (IFNγ) and inducible nitric oxide synthase (iNOS)
Modulation of inducible nitric oxide synthase expression by sumoylation
Monocytes differentiate along two alternative pathways during sterile inflammation
Malarial pigment haemozoin, IFN-gamma, TNF-alpha, IL-1beta and LPS do not stimulate expression of inducible nitric oxide synthase and production of nitric oxide in immuno-purified human monocytes
Type I interferon supports inducible nitric oxide synthase in murine hepatoma cells and hepatocytes and during experimental acetaminophen-induced liver damage
P2Y2 impacts inflammatory monocytes during acute Graft-versus-Host Disease
The role of nitric oxide during embryonic wound healing
The Role of Inducible Nitric Oxide Synthase (iNOS) in Human Myocardial Infarction
Tissue expression and cytokine regulation of the human inducible nitric oxide synthase (iNOS)
Inducible nitric oxide synthase inhibits macrophage migration, a potential explanation for iNOS's proatherosclerotic action
Inducible Nitric Oxide Synthase inhibits macrophage migration,a potential explanation for iNOS's proatherosclerotic action
Inducible Nitric Oxide Synthase (iNOS) Is a Novel Negative Regulator of Hematopoietic Stem/Progenitor Cell Trafficking
Reproducibility and intraindividual variation over days in buccal cell DNA methylation of two asthma genes, interferon γ (IFNγ) and inducible nitric oxide synthase (iNOS)
Modulation of inducible nitric oxide synthase expression by sumoylation
Monocytes differentiate along two alternative pathways during sterile inflammation
Malarial pigment haemozoin, IFN-gamma, TNF-alpha, IL-1beta and LPS do not stimulate expression of inducible nitric oxide synthase and production of nitric oxide in immuno-purified human monocytes
Type I interferon supports inducible nitric oxide synthase in murine hepatoma cells and hepatocytes and during experimental acetaminophen-induced liver damage
P2Y2 impacts inflammatory monocytes during acute Graft-versus-Host Disease