The expression and regulation of matrix metalloproteinase-3 is critically modulated by Porphyromonas gingivalis lipopolysaccharide with heterogeneous lipid A structures in human gingival fibroblasts
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2180
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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The expression and regulation of matrix metalloproteinase-3 is critically modulated by Porphyromonas gingivalis lipopolysaccharide with heterogeneous lipid A structures in human gingival fibroblasts ; volume:13 ; number:1 ; day:30 ; month:3 ; year:2013 ; pages:1-12 ; date:12.2013
BMC microbiology ; 13, Heft 1 (30.3.2013), 1-12, 12.2013
- Creator
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Herath, Thanuja D K.
- Contributor
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Wang, Yu
Seneviratne, Chaminda J.
Darveau, Richard P.
Wang, Cun-Yu
Jin, Lijian
SpringerLink (Online service)
- DOI
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10.1186/1471-2180-13-73
- URN
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urn:nbn:de:1111-2016030238576
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:38 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Herath, Thanuja D K.
- Wang, Yu
- Seneviratne, Chaminda J.
- Darveau, Richard P.
- Wang, Cun-Yu
- Jin, Lijian
- SpringerLink (Online service)
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Hexokinase 2-mediated glycolysis supports inflammatory responses to Porphyromonas gingivalis in gingival fibroblasts
The capsule of Porphyromonas gingivalis reduces the immune response of human gingival fibroblasts
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TNF-α augmented Porphyromonas gingivalis invasion in human gingival epithelial cells through Rab5 and ICAM-1
Commensal Neisseria Inhibit Porphyromonas Gingivalis Invasion of Gingival Epithelial Cells
HSP90AA1 promotes the inflammation in human gingival fibroblasts induced by Porphyromonas gingivalis lipopolysaccharide via regulating of autophagy
The promotion of nephropathy by Porphyromonas gingivalis lipopolysaccharide via toll-like receptors
Hexokinase 2-mediated glycolysis supports inflammatory responses to Porphyromonas gingivalis in gingival fibroblasts
The capsule of Porphyromonas gingivalis reduces the immune response of human gingival fibroblasts
Effects of Porphyromonas gingivalis Lipopolysaccharide on human dental follicle progenitor cells in vitro
Effects of Porphyromonas gingivalis Lipopolysaccharide on human dental follicle progenitor cells in vitro
Methanandamide diminish the Porphyromonas gingivalis lipopolysaccharide induced response in human periodontal ligament cells
Modulation of protease-activated receptor expression by Porphyromonas gingivalis in human gingival epithelial cells
Porphyromonas gingivalis induce apoptosis in human gingival epithelial cells through a gingipain-dependent mechanism
Vidarabine, an anti-herpes agent, improves Porphyromonas gingivalis lipopolysaccharide-induced cardiac dysfunction in mice
TNF-α augmented Porphyromonas gingivalis invasion in human gingival epithelial cells through Rab5 and ICAM-1
Commensal Neisseria Inhibit Porphyromonas Gingivalis Invasion of Gingival Epithelial Cells
HSP90AA1 promotes the inflammation in human gingival fibroblasts induced by Porphyromonas gingivalis lipopolysaccharide via regulating of autophagy
The promotion of nephropathy by Porphyromonas gingivalis lipopolysaccharide via toll-like receptors
Hexokinase 2-mediated glycolysis supports inflammatory responses to Porphyromonas gingivalis in gingival fibroblasts
The capsule of Porphyromonas gingivalis reduces the immune response of human gingival fibroblasts
Effects of Porphyromonas gingivalis Lipopolysaccharide on human dental follicle progenitor cells in vitro
Effects of Porphyromonas gingivalis Lipopolysaccharide on human dental follicle progenitor cells in vitro
Methanandamide diminish the Porphyromonas gingivalis lipopolysaccharide induced response in human periodontal ligament cells
Modulation of protease-activated receptor expression by Porphyromonas gingivalis in human gingival epithelial cells
Porphyromonas gingivalis induce apoptosis in human gingival epithelial cells through a gingipain-dependent mechanism
Vidarabine, an anti-herpes agent, improves Porphyromonas gingivalis lipopolysaccharide-induced cardiac dysfunction in mice