ALS mutant SOD1 interacts with G3BP1 and affects stress granule dynamics

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1432-0533

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: ALS mutant SOD1 interacts with G3BP1 and affects stress granule dynamics ; day:1 ; month:8 ; year:2016 ; pages:1-14
Acta neuropathologica ; (1.8.2016), 1-14

Creator: Gál, József

Contributor: Kuang, Lisha
Barnett, Kelly R.
Zhu, Brian Z.
Shissler, Susannah C.
Korotkov, Konstantin V.
Hayward, Lawrence J.
Kasarskis, Edward J.
Zhu, Haining
SpringerLink (Online service)

DOI: 10.1007/s00401-016-1601-x

URN: urn:nbn:de:1111-201608034978

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:47 AM CEST

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Associated

Gál, József
Kuang, Lisha
Barnett, Kelly R.
Zhu, Brian Z.
Shissler, Susannah C.
Korotkov, Konstantin V.
Hayward, Lawrence J.
Kasarskis, Edward J.
Zhu, Haining
SpringerLink (Online service)

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ALS mutant SOD1 interacts with G3BP1 and affects stress granule dynamics

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Other Objects (12)

Better survival in female SOD1-mutant patients with ALS: a study of SOD1-related natural history

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Impaired ATF3 signaling involves SNAP25 in SOD1 mutant ALS patients

Correction to: Better survival in female SOD1-mutant patients with ALS: a study of SOD1-related natural history

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Boundary Cap Neural Crest Stem Cells Promote Survival of Mutant SOD1 Motor Neurons

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SOD1-positive aggregate accumulation in the CNS predicts slower disease progression and increased longevity in a mutant SOD1 mouse model of ALS

Stathmin 1/2-triggered microtubule loss mediates Golgi fragmentation in mutant SOD1 motor neurons

Differential induction of mutant SOD1 misfolding and aggregation by tau and α-synuclein pathology

PV1, a novel Plasmodium falciparum merozoite dense granule protein, interacts with exported protein in infected erythrocytes

ZO-1 interacts with YB-1 in endothelial cells to regulate stress granule formation during angiogenesis

Better survival in female SOD1-mutant patients with ALS: a study of SOD1-related natural history

Increased surface P2X4 receptors by mutant SOD1 proteins contribute to ALS pathogenesis in SOD1-G93A mice

Impaired ATF3 signaling involves SNAP25 in SOD1 mutant ALS patients

Correction to: Better survival in female SOD1-mutant patients with ALS: a study of SOD1-related natural history

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Boundary Cap Neural Crest Stem Cells Promote Survival of Mutant SOD1 Motor Neurons

Intracellular spatially-targeted chemical chaperones increase native state stability of mutant SOD1 barrel

SOD1-positive aggregate accumulation in the CNS predicts slower disease progression and increased longevity in a mutant SOD1 mouse model of ALS

Stathmin 1/2-triggered microtubule loss mediates Golgi fragmentation in mutant SOD1 motor neurons

Differential induction of mutant SOD1 misfolding and aggregation by tau and α-synuclein pathology

PV1, a novel Plasmodium falciparum merozoite dense granule protein, interacts with exported protein in infected erythrocytes

ZO-1 interacts with YB-1 in endothelial cells to regulate stress granule formation during angiogenesis

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Increased surface P2X4 receptors by mutant SOD1 proteins contribute to ALS pathogenesis in SOD1-G93A mice

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Correction to: Better survival in female SOD1-mutant patients with ALS: a study of SOD1-related natural history

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PV1, a novel Plasmodium falciparum merozoite dense granule protein, interacts with exported protein in infected erythrocytes

ZO-1 interacts with YB-1 in endothelial cells to regulate stress granule formation during angiogenesis

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