Hochschulschrift
Interplay of Plk1 inhibition with inhibition of further important cancer targets
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Dimensions
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30 cm
- Extent
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49 Seiten
- Language
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Englisch
- Notes
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Illustrationen, Diagramme
Enthält Beiträge aus verschiedenen Zeitschriften
Friedrich-Schiller-Universität Jena, Dissertation, 2016
- Classification
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Medizin, Gesundheit
- Keyword
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Kinasen
Inhibitor
Krebsforschung
Zelllinie
- Table of contents
- Rights
-
Bei diesem Objekt liegt nur das Inhaltsverzeichnis digital vor. Der Zugriff darauf ist unbeschränkt möglich.
- Last update
-
11.06.2025, 1:35 PM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Object type
- Hochschulschrift
Associated
Time of origin
- [2016]
Other Objects (12)
Therapeutic potential of PLK1 inhibition in triple-negative breast cancer
PLK1 overexpression suppresses homologous recombination and confers cellular sensitivity to PARP inhibition
Inhibition of PLK1-dependent phosphorylation of EBNA2 promotes EBV-induced carcinogenesis in humanized mice
Targeting Echinococcus multilocularis Stem Cells by Inhibition of the Polo-Like Kinase EmPlk1
Further important extracts
Inhibition of the PLK1‐Coupled Cell Cycle Machinery Overcomes Resistance to Oxaliplatin in Colorectal Cancer
Polo-like kinase 1 (PLK1) inhibition suppresses cell growth and enhances radiation sensitivity in medulloblastoma cells
Interplay between periodic stimulation and GABAergic inhibition in striatal network oscillations
Further study of the inhibition of premature labor by indomethacin. Part I
Inhibition von PLK1 als therapeutische Strategie für Patienten mit myelodysplastischen Syndromen (MDS) und sekundären akuten myeloischen Leukämien (sAML)
Proteinasen-Inhibition
Inhibition, Limulus
Therapeutic potential of PLK1 inhibition in triple-negative breast cancer
PLK1 overexpression suppresses homologous recombination and confers cellular sensitivity to PARP inhibition
Inhibition of PLK1-dependent phosphorylation of EBNA2 promotes EBV-induced carcinogenesis in humanized mice
Targeting Echinococcus multilocularis Stem Cells by Inhibition of the Polo-Like Kinase EmPlk1
Further important extracts
Inhibition of the PLK1‐Coupled Cell Cycle Machinery Overcomes Resistance to Oxaliplatin in Colorectal Cancer
Polo-like kinase 1 (PLK1) inhibition suppresses cell growth and enhances radiation sensitivity in medulloblastoma cells
Interplay between periodic stimulation and GABAergic inhibition in striatal network oscillations
Further study of the inhibition of premature labor by indomethacin. Part I
Inhibition von PLK1 als therapeutische Strategie für Patienten mit myelodysplastischen Syndromen (MDS) und sekundären akuten myeloischen Leukämien (sAML)
Proteinasen-Inhibition
Inhibition, Limulus
Therapeutic potential of PLK1 inhibition in triple-negative breast cancer
PLK1 overexpression suppresses homologous recombination and confers cellular sensitivity to PARP inhibition
Inhibition of PLK1-dependent phosphorylation of EBNA2 promotes EBV-induced carcinogenesis in humanized mice
Targeting Echinococcus multilocularis Stem Cells by Inhibition of the Polo-Like Kinase EmPlk1
Further important extracts
Inhibition of the PLK1‐Coupled Cell Cycle Machinery Overcomes Resistance to Oxaliplatin in Colorectal Cancer
Polo-like kinase 1 (PLK1) inhibition suppresses cell growth and enhances radiation sensitivity in medulloblastoma cells
Interplay between periodic stimulation and GABAergic inhibition in striatal network oscillations
Further study of the inhibition of premature labor by indomethacin. Part I
Inhibition von PLK1 als therapeutische Strategie für Patienten mit myelodysplastischen Syndromen (MDS) und sekundären akuten myeloischen Leukämien (sAML)
Proteinasen-Inhibition