Reversible inactivation of the transcriptional function of P53 protein by farnesylation
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1472-6750
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
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online resource.
- Erschienen in
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Reversible inactivation of the transcriptional function of P53 protein by farnesylation ; volume:6 ; number:1 ; day:29 ; month:5 ; year:2006 ; pages:1-11 ; date:12.2006
BMC biotechnology ; 6, Heft 1 (29.5.2006), 1-11, 12.2006
- Urheber
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Couderc, Bettina
- Beteiligte Personen und Organisationen
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Penary, Marie
Tohfe, Mustapha
Pradines, Anne
Casteignau, Antoine
Berg, Danièle
Favre, Gilles
SpringerLink (Online service)
- DOI
-
10.1186/1472-6750-6-26
- URN
-
urn:nbn:de:1111-2016072532222
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:52 MESZ
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Beteiligte
- Couderc, Bettina
- Penary, Marie
- Tohfe, Mustapha
- Pradines, Anne
- Casteignau, Antoine
- Berg, Danièle
- Favre, Gilles
- SpringerLink (Online service)
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FBXW7 inactivation induces cellular senescence via accumulation of p53
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Role of p53 in transcriptional regulation of proteases in mammals.
Impact of p53 inactivation in genomic instability during DNA damage induced senescence
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NGF-mediated transcriptional targets of p53 in PC12 neuronal differentiation
Interaction of p53 with the Δ133p53α and Δ160p53α isoforms regulates p53 conformation and transcriptional activity
Transglutaminase 2 promotes tumorigenicity of colon cancer cells by inactivation of the tumor suppressor p53
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Nuclear p53-mediated repression of autophagy involves PINK1 transcriptional down-regulation
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Reversible Inactivation of the Nitrate Reductase of Chlorella Vulgaris Beijerinck
FBXW7 inactivation induces cellular senescence via accumulation of p53
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Role of p53 in transcriptional regulation of proteases in mammals.
Impact of p53 inactivation in genomic instability during DNA damage induced senescence
Structural basis of p53 inactivation by cavity-creating cancer mutations and its implications for the development of mutant p53 reactivators
NGF-mediated transcriptional targets of p53 in PC12 neuronal differentiation
Interaction of p53 with the Δ133p53α and Δ160p53α isoforms regulates p53 conformation and transcriptional activity
Transglutaminase 2 promotes tumorigenicity of colon cancer cells by inactivation of the tumor suppressor p53
p63, p53 wild type and UV induced p53 mutants influence the transcriptional activities of cutaneous HPV
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