BRCA 1 and Ct IP promote alternative non‐homologous end‐joining at uncapped telomeres

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: BRCA 1 and Ct IP promote alternative non‐homologous end‐joining at uncapped telomeres ; volume:34 ; number:3 ; year:2015 ; pages:410-424 ; extent:15
The EMBO journal / European Molecular Biology Organization ; 34, Heft 3 (2015), 410-424 (gesamt 15)

Creator: Badie, Sophie
Carlos, Ana Rita
Folio, Cecilia
Okamoto, Keiji
Bouwman, Peter
Jonkers, Jos
Tarsounas, Madalena

DOI: 10.15252/embj.201488947

URN: urn:nbn:de:101:1-2022112407503017154838

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:24 AM CEST

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Badie, Sophie
Carlos, Ana Rita
Folio, Cecilia
Okamoto, Keiji
Bouwman, Peter
Jonkers, Jos
Tarsounas, Madalena

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Resection-dependent canonical non-homologous end-joining induces genomic rearrangements

OTUD5 promotes end-joining of deprotected telomeres by promoting ATM-dependent phosphorylation of KAP1S824

One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks

Mosaic mitochondrial-plastid insertions into the nuclear genome show evidence of both non-homologous end joining and homologous recombination

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Merotelic attachments and non-homologous end joining are the basis of chromosomal instability

SIRT 7 promotes genome integrity and modulates non‐homologous end joining DNA repair

BRCA loss of function including BRCA1 DNA-methylation, but not BRCA-unrelated homologous recombination deficiency, is associated with platinum hypersensitivity in high-grade ovarian cancer

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

CtIP and MRN promote non-homologous end-joining of etoposide-induced DNA double-strand breaks in G1

DNA non-homologous end-joining enters the resection arena

PHF6 promotes non‐homologous end joining and G2 checkpoint recovery

Resection-dependent canonical non-homologous end-joining induces genomic rearrangements

OTUD5 promotes end-joining of deprotected telomeres by promoting ATM-dependent phosphorylation of KAP1S824

One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks

Mosaic mitochondrial-plastid insertions into the nuclear genome show evidence of both non-homologous end joining and homologous recombination

Retrotransposons and Telomeres

Merotelic attachments and non-homologous end joining are the basis of chromosomal instability

SIRT 7 promotes genome integrity and modulates non‐homologous end joining DNA repair

BRCA loss of function including BRCA1 DNA-methylation, but not BRCA-unrelated homologous recombination deficiency, is associated with platinum hypersensitivity in high-grade ovarian cancer

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

CtIP and MRN promote non-homologous end-joining of etoposide-induced DNA double-strand breaks in G1

DNA non-homologous end-joining enters the resection arena

PHF6 promotes non‐homologous end joining and G2 checkpoint recovery

Resection-dependent canonical non-homologous end-joining induces genomic rearrangements

OTUD5 promotes end-joining of deprotected telomeres by promoting ATM-dependent phosphorylation of KAP1S824

One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks

Mosaic mitochondrial-plastid insertions into the nuclear genome show evidence of both non-homologous end joining and homologous recombination

Retrotransposons and Telomeres

Merotelic attachments and non-homologous end joining are the basis of chromosomal instability

SIRT 7 promotes genome integrity and modulates non‐homologous end joining DNA repair

BRCA loss of function including BRCA1 DNA-methylation, but not BRCA-unrelated homologous recombination deficiency, is associated with platinum hypersensitivity in high-grade ovarian cancer

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

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BRCA 1 and Ct IP promote alternative non‐homologous end‐joining at uncapped telomeres

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Other Objects (12)

CtIP and MRN promote non-homologous end-joining of etoposide-induced DNA double-strand breaks in G1

DNA non-homologous end-joining enters the resection arena

PHF6 promotes non‐homologous end joining and G2 checkpoint recovery

Resection-dependent canonical non-homologous end-joining induces genomic rearrangements

OTUD5 promotes end-joining of deprotected telomeres by promoting ATM-dependent phosphorylation of KAP1S824

One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks

Mosaic mitochondrial-plastid insertions into the nuclear genome show evidence of both non-homologous end joining and homologous recombination

Retrotransposons and Telomeres

Merotelic attachments and non-homologous end joining are the basis of chromosomal instability

SIRT 7 promotes genome integrity and modulates non‐homologous end joining DNA repair

BRCA loss of function including BRCA1 DNA-methylation, but not BRCA-unrelated homologous recombination deficiency, is associated with platinum hypersensitivity in high-grade ovarian cancer

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

CtIP and MRN promote non-homologous end-joining of etoposide-induced DNA double-strand breaks in G1

DNA non-homologous end-joining enters the resection arena

PHF6 promotes non‐homologous end joining and G2 checkpoint recovery

Resection-dependent canonical non-homologous end-joining induces genomic rearrangements

OTUD5 promotes end-joining of deprotected telomeres by promoting ATM-dependent phosphorylation of KAP1S824

One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks

Mosaic mitochondrial-plastid insertions into the nuclear genome show evidence of both non-homologous end joining and homologous recombination

Retrotransposons and Telomeres

Merotelic attachments and non-homologous end joining are the basis of chromosomal instability

SIRT 7 promotes genome integrity and modulates non‐homologous end joining DNA repair

BRCA loss of function including BRCA1 DNA-methylation, but not BRCA-unrelated homologous recombination deficiency, is associated with platinum hypersensitivity in high-grade ovarian cancer

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

CtIP and MRN promote non-homologous end-joining of etoposide-induced DNA double-strand breaks in G1

DNA non-homologous end-joining enters the resection arena

PHF6 promotes non‐homologous end joining and G2 checkpoint recovery

Resection-dependent canonical non-homologous end-joining induces genomic rearrangements

OTUD5 promotes end-joining of deprotected telomeres by promoting ATM-dependent phosphorylation of KAP1S824

One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks

Mosaic mitochondrial-plastid insertions into the nuclear genome show evidence of both non-homologous end joining and homologous recombination

Retrotransposons and Telomeres

Merotelic attachments and non-homologous end joining are the basis of chromosomal instability

SIRT 7 promotes genome integrity and modulates non‐homologous end joining DNA repair

BRCA loss of function including BRCA1 DNA-methylation, but not BRCA-unrelated homologous recombination deficiency, is associated with platinum hypersensitivity in high-grade ovarian cancer

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

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