Hochschulschrift
Pharmacological preconditioning of the rat heart by Xenon and Isoflurane : involvement of PKCe, p38 MAPK and Hsp 27
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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Düsseldorf, Univ., Diss., 2006
- Creator
- URN
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urn:nbn:de:hbz:061-20070227-090039-0
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:49 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Object type
- Hochschulschrift
Associated
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RhoA/rho-kinase, nitric oxide and inflammatory response in LIMA during OPCABG with isoflurane preconditioning
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Human HspB1, HspB3, HspB5 and HspB8: Shaping these disease factors during vertebrate evolution
Werkstatt Hsp.
Isoflurane Preconditioning at Clinically Relevant Doses Induce Protective Effects of Heme Oxygenase-1 on Hepatic Ischemia Reperfusion in Rats
The effect of isoflurane and xenon on the synapse elimination by astrocytes in the absent/present of Aβ1-42
Acidic preconditioning protects endothelial cells against apoptosis through p38- and Akt-dependent Bcl-xL overexpression
Xenon
Xenon preconditioning: molecular mechanisms and biological effects
Isoflurane
Intravenous pretreatment with emulsified isoflurane preconditioning protects kidneys against ischemia/reperfusion injury in rats
RhoA/rho-kinase, nitric oxide and inflammatory response in LIMA during OPCABG with isoflurane preconditioning
Repeated remote ischemic preconditioning and isoflurane anesthesia in an experimental model of renal ischemia-reperfusion injury
Recovery index, attentiveness and state of memory after xenon or isoflurane anaesthesia: a randomized controlled trial
Human HspB1, HspB3, HspB5 and HspB8: Shaping these disease factors during vertebrate evolution
Werkstatt Hsp.
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The effect of isoflurane and xenon on the synapse elimination by astrocytes in the absent/present of Aβ1-42
Acidic preconditioning protects endothelial cells against apoptosis through p38- and Akt-dependent Bcl-xL overexpression