- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1746-1596
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
GATA4 and DcR1 methylation in glioblastomas ; volume:8 ; number:1 ; day:15 ; month:1 ; year:2013 ; pages:1-5 ; date:12.2013
Diagnostic pathology ; 8, Heft 1 (15.1.2013), 1-5, 12.2013
- Creator
-
Vaitkiene, Paulina
- Contributor
-
Skiriute, Daina
Skauminas, Kestutis
Tamasauskas, Arimantas
SpringerLink (Online service)
- DOI
-
10.1186/1746-1596-8-7
- URN
-
urn:nbn:de:1111-2016080744061
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:53 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Vaitkiene, Paulina
- Skiriute, Daina
- Skauminas, Kestutis
- Tamasauskas, Arimantas
- SpringerLink (Online service)
Other Objects (12)
Stem cell phenotype predicts therapeutic response in glioblastomas with MGMT promoter methylation
MGMT methylation may benefit overall survival in patients with moderately vascularized glioblastomas
Heterogeneity of DNA methylation profiles and copy number alterations in 10782 adult-type glioblastomas, IDH-wildtype
A combination of TERT promoter mutation and MGMT methylation status predicts clinically relevant subgroups of newly diagnosed glioblastomas
MGMT gene promoter methylation by pyrosequencing method correlates volumetric response and neurological status in IDH wild-type glioblastomas
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Somatic retrotransposition is infrequent in glioblastomas
Glioblastomas with primitive neuronal component harbor a distinct methylation and copy-number profile with inactivation of TP53, PTEN, and RB1
Glioblastomas with primitive neuronal component harbor a distinct methylation and copy-number profile with inactivation of TP53, PTEN, and RB1
Stem cell phenotype predicts therapeutic response in glioblastomas with MGMT promoter methylation
MGMT methylation may benefit overall survival in patients with moderately vascularized glioblastomas
Heterogeneity of DNA methylation profiles and copy number alterations in 10782 adult-type glioblastomas, IDH-wildtype
A combination of TERT promoter mutation and MGMT methylation status predicts clinically relevant subgroups of newly diagnosed glioblastomas
MGMT gene promoter methylation by pyrosequencing method correlates volumetric response and neurological status in IDH wild-type glioblastomas
R1, 4 R1, 5 R1, 6
R1, 1 R1, 2 R1, 3
R1, 7 R1, 8 R1, 9
R1, 10 R1, 11 R1, 12
Somatic retrotransposition is infrequent in glioblastomas
Glioblastomas with primitive neuronal component harbor a distinct methylation and copy-number profile with inactivation of TP53, PTEN, and RB1
Glioblastomas with primitive neuronal component harbor a distinct methylation and copy-number profile with inactivation of TP53, PTEN, and RB1
Stem cell phenotype predicts therapeutic response in glioblastomas with MGMT promoter methylation
MGMT methylation may benefit overall survival in patients with moderately vascularized glioblastomas
Heterogeneity of DNA methylation profiles and copy number alterations in 10782 adult-type glioblastomas, IDH-wildtype
A combination of TERT promoter mutation and MGMT methylation status predicts clinically relevant subgroups of newly diagnosed glioblastomas
MGMT gene promoter methylation by pyrosequencing method correlates volumetric response and neurological status in IDH wild-type glioblastomas
R1, 4 R1, 5 R1, 6
R1, 1 R1, 2 R1, 3
R1, 7 R1, 8 R1, 9
R1, 10 R1, 11 R1, 12
Somatic retrotransposition is infrequent in glioblastomas
Glioblastomas with primitive neuronal component harbor a distinct methylation and copy-number profile with inactivation of TP53, PTEN, and RB1