Hochschulschrift
A high-complexity lentiviral shRNA screen identifies synthetic lethal interactions with deregulated N-Myc in neuroblastoma cells
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Dimensions
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30 cm
- Extent
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VII, 192 S.
- Language
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Englisch
- Notes
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Ill., graph. Darst.
Würzburg, Univ., Diss., 2014
- Classification
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Medizin, Gesundheit
Biowissenschaften, Biologie
- Creator
- Table of contents
- Rights
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Bei diesem Objekt liegt nur das Inhaltsverzeichnis digital vor. Der Zugriff darauf ist unbeschränkt möglich.
- Last update
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11.06.2025, 1:44 PM CEST
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Object type
- Hochschulschrift
Associated
Other Objects (12)
A high-complexity lentiviral shRNA screen identifies synthetic lethal interactions with deregulated N-Myc in neuroblastoma cells
Decoding the metabolic program of deregulated MYCN expression in neuroblastoma
A Selective Small‐Molecule c‐Myc Degrader Potently Regresses Lethal c‐Myc Overexpressing Tumors
Deregulated expression of microRNAs : a critical pathogenic mechanism in MYC-induced lymphoma
Mitoribosomal synthetic lethality overcomes multidrug resistance in MYC-driven neuroblastoma
N-Myc-induced metabolic rewiring creates novel therapeutic vulnerabilities in neuroblastoma
Extracellular matrix rigidity modulates neuroblastoma cell differentiation and N-myc expression
MYC and the unfolded protein response in cancer: synthetic lethal partners in crime?
The lethal giant larvaetumour suppressor mutation requires dMyc oncoprotein to promote clonal malignancy
Genetic screening for anticancer genes highlights FBLN5 as a synthetic lethal partner of MYC
The initial mechanism involved in N-myc oncogene amplification in human neuroblastoma cell lines
Transcription factor activating protein 4 is synthetically lethal and a master regulator of MYCN-amplified neuroblastoma
A high-complexity lentiviral shRNA screen identifies synthetic lethal interactions with deregulated N-Myc in neuroblastoma cells
Decoding the metabolic program of deregulated MYCN expression in neuroblastoma
A Selective Small‐Molecule c‐Myc Degrader Potently Regresses Lethal c‐Myc Overexpressing Tumors
Deregulated expression of microRNAs : a critical pathogenic mechanism in MYC-induced lymphoma
Mitoribosomal synthetic lethality overcomes multidrug resistance in MYC-driven neuroblastoma
N-Myc-induced metabolic rewiring creates novel therapeutic vulnerabilities in neuroblastoma
Extracellular matrix rigidity modulates neuroblastoma cell differentiation and N-myc expression
MYC and the unfolded protein response in cancer: synthetic lethal partners in crime?
The lethal giant larvaetumour suppressor mutation requires dMyc oncoprotein to promote clonal malignancy
Genetic screening for anticancer genes highlights FBLN5 as a synthetic lethal partner of MYC
The initial mechanism involved in N-myc oncogene amplification in human neuroblastoma cell lines
Transcription factor activating protein 4 is synthetically lethal and a master regulator of MYCN-amplified neuroblastoma
A high-complexity lentiviral shRNA screen identifies synthetic lethal interactions with deregulated N-Myc in neuroblastoma cells
Decoding the metabolic program of deregulated MYCN expression in neuroblastoma
A Selective Small‐Molecule c‐Myc Degrader Potently Regresses Lethal c‐Myc Overexpressing Tumors
Deregulated expression of microRNAs : a critical pathogenic mechanism in MYC-induced lymphoma
Mitoribosomal synthetic lethality overcomes multidrug resistance in MYC-driven neuroblastoma
N-Myc-induced metabolic rewiring creates novel therapeutic vulnerabilities in neuroblastoma
Extracellular matrix rigidity modulates neuroblastoma cell differentiation and N-myc expression
MYC and the unfolded protein response in cancer: synthetic lethal partners in crime?
The lethal giant larvaetumour suppressor mutation requires dMyc oncoprotein to promote clonal malignancy
Genetic screening for anticancer genes highlights FBLN5 as a synthetic lethal partner of MYC
The initial mechanism involved in N-myc oncogene amplification in human neuroblastoma cell lines