FLIP(C1orf112)-FIGNL1 complex regulates RAD51 chromatin association to promote viability after replication stress
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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FLIP(C1orf112)-FIGNL1 complex regulates RAD51 chromatin association to promote viability after replication stress ; volume:15 ; number:1 ; day:29 ; month:1 ; year:2024 ; pages:1-18 ; date:12.2024
Nature Communications ; 15, Heft 1 (29.1.2024), 1-18, 12.2024
- Creator
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Tischler, Jessica D.
Tsuchida, Hiroshi
Bosire, Rosevalentine
Oda, Tommy T.
Park, Ana
Adeyemi, Richard O.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41467-024-45139-9
- URN
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urn:nbn:de:101:1-2024040910264012291650
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:46 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Tischler, Jessica D.
- Tsuchida, Hiroshi
- Bosire, Rosevalentine
- Oda, Tommy T.
- Park, Ana
- Adeyemi, Richard O.
- SpringerLink (Online service)
Other Objects (12)
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Sister chromatid exchanges induced by perturbed replication can form independently of BRCA1, BRCA2 and RAD51
PARP1 and PARP2 stabilise replication forks at base excision repair intermediates through Fbh1-dependent Rad51 regulation
FIGNL1 AAA+ ATPase remodels RAD51 and DMC1 filaments in pre-meiotic DNA replication and meiotic recombination
Homocysteine affects cardiomyocyte viability: concentration-dependent effects on reversible flip-flop, apoptosis and necrosis
The replication initiation determinant protein (RepID) modulates replication by recruiting CUL4 to chromatin
"FLIP"
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Interplay of DNA replication, repair and chromatin: structure versus function
Interplay of DNA replication, repair and chromatin under radiation stress
Chromatin-dependent pre-replication complex positioning and activation in mammals
Loss of SIM2s inhibits RAD51 binding and leads to unresolved replication stress
GRB2 stabilizes RAD51 at reversed replication forks suppressing genomic instability and innate immunity against cancer
Sister chromatid exchanges induced by perturbed replication can form independently of BRCA1, BRCA2 and RAD51
PARP1 and PARP2 stabilise replication forks at base excision repair intermediates through Fbh1-dependent Rad51 regulation
FIGNL1 AAA+ ATPase remodels RAD51 and DMC1 filaments in pre-meiotic DNA replication and meiotic recombination
Homocysteine affects cardiomyocyte viability: concentration-dependent effects on reversible flip-flop, apoptosis and necrosis
The replication initiation determinant protein (RepID) modulates replication by recruiting CUL4 to chromatin
"FLIP"
FLIP
Interplay of DNA replication, repair and chromatin: structure versus function
Interplay of DNA replication, repair and chromatin under radiation stress
Chromatin-dependent pre-replication complex positioning and activation in mammals
Loss of SIM2s inhibits RAD51 binding and leads to unresolved replication stress
GRB2 stabilizes RAD51 at reversed replication forks suppressing genomic instability and innate immunity against cancer
Sister chromatid exchanges induced by perturbed replication can form independently of BRCA1, BRCA2 and RAD51
PARP1 and PARP2 stabilise replication forks at base excision repair intermediates through Fbh1-dependent Rad51 regulation
FIGNL1 AAA+ ATPase remodels RAD51 and DMC1 filaments in pre-meiotic DNA replication and meiotic recombination
Homocysteine affects cardiomyocyte viability: concentration-dependent effects on reversible flip-flop, apoptosis and necrosis
The replication initiation determinant protein (RepID) modulates replication by recruiting CUL4 to chromatin
"FLIP"
FLIP
Interplay of DNA replication, repair and chromatin: structure versus function
Interplay of DNA replication, repair and chromatin under radiation stress