Human cytomegalovirus exploits STING signaling and counteracts IFN/ISG induction to facilitate infection of dendritic cells

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: Human cytomegalovirus exploits STING signaling and counteracts IFN/ISG induction to facilitate infection of dendritic cells ; volume:15 ; number:1 ; day:26 ; month:2 ; year:2024 ; pages:1-16 ; date:12.2024
Nature Communications ; 15, Heft 1 (26.2.2024), 1-16, 12.2024

Creator: Costa, Bibiana
Becker, Jennifer
Krammer, Tobias
Mulenge, Felix
Durán Mejía, Verónica
Pavlou, Andreas
Gern, Olivia Luise
Chu, Xiaojing
Li, Yang
Čičin-Šain, Luka
Eiz-Vesper, Britta
Messerle, Martin
Dölken, Lars
Saliba, Antoine-Emmanuel
Erhard, Florian
Kalinke, Ulrich

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41467-024-45614-3

URN: urn:nbn:de:101:1-2405021315037.876696181347

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:51 AM CEST

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Associated

Costa, Bibiana
Becker, Jennifer
Krammer, Tobias
Mulenge, Felix
Durán Mejía, Verónica
Pavlou, Andreas
Gern, Olivia Luise
Chu, Xiaojing
Li, Yang
Čičin-Šain, Luka
Eiz-Vesper, Britta
Messerle, Martin
Dölken, Lars
Saliba, Antoine-Emmanuel
Erhard, Florian
Kalinke, Ulrich
SpringerLink (Online service)

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STING induces early IFN-beta in the liver and constrains myeloid cell-mediated dissemination of murine cytomegalovirus

Influenza C virus NS1 protein counteracts RIG-I-mediated IFN signalling

DUSP4 modulates RIG-I- and STING-mediated IRF3-type I IFN response

TLR9 and STING agonists synergistically induce innate and adaptive type‐II IFN

ARF1 controls STING activation and recycling to prevent chronic type I IFN signalling

PRV UL13 inhibits cGAS–STING-mediated IFN-β production by phosphorylating IRF3

Exploits des François

Aryl hydrocarbon receptor suppresses STING-mediated type I IFN expression in triple-negative breast cancer

Monocyte-derived macrophages aggravate pulmonary vasculitis via cGAS/STING/IFN-mediated nucleic acid sensing

SGLT2 inhibitor activates the STING/IRF3/IFN-β pathway and induces immune infiltration in osteosarcoma

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STING induces early IFN-beta in the liver and constrains myeloid cell-mediated dissemination of murine cytomegalovirus

Influenza C virus NS1 protein counteracts RIG-I-mediated IFN signalling

DUSP4 modulates RIG-I- and STING-mediated IRF3-type I IFN response

TLR9 and STING agonists synergistically induce innate and adaptive type‐II IFN

ARF1 controls STING activation and recycling to prevent chronic type I IFN signalling

PRV UL13 inhibits cGAS–STING-mediated IFN-β production by phosphorylating IRF3

Exploits des François

Aryl hydrocarbon receptor suppresses STING-mediated type I IFN expression in triple-negative breast cancer

Monocyte-derived macrophages aggravate pulmonary vasculitis via cGAS/STING/IFN-mediated nucleic acid sensing

SGLT2 inhibitor activates the STING/IRF3/IFN-β pathway and induces immune infiltration in osteosarcoma

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Human cytomegalovirus exploits STING signaling and counteracts IFN/ISG induction to facilitate infection of dendritic cells

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Other Objects (12)

STING induces early IFN-beta in the liver and constrains myeloid cell-mediated dissemination of murine cytomegalovirus

Influenza C virus NS1 protein counteracts RIG-I-mediated IFN signalling

DUSP4 modulates RIG-I- and STING-mediated IRF3-type I IFN response

TLR9 and STING agonists synergistically induce innate and adaptive type‐II IFN

ARF1 controls STING activation and recycling to prevent chronic type I IFN signalling

PRV UL13 inhibits cGAS–STING-mediated IFN-β production by phosphorylating IRF3

Exploits des François

Aryl hydrocarbon receptor suppresses STING-mediated type I IFN expression in triple-negative breast cancer

Monocyte-derived macrophages aggravate pulmonary vasculitis via cGAS/STING/IFN-mediated nucleic acid sensing

SGLT2 inhibitor activates the STING/IRF3/IFN-β pathway and induces immune infiltration in osteosarcoma

Sting

Sting

STING induces early IFN-beta in the liver and constrains myeloid cell-mediated dissemination of murine cytomegalovirus

Influenza C virus NS1 protein counteracts RIG-I-mediated IFN signalling

DUSP4 modulates RIG-I- and STING-mediated IRF3-type I IFN response

TLR9 and STING agonists synergistically induce innate and adaptive type‐II IFN

ARF1 controls STING activation and recycling to prevent chronic type I IFN signalling

PRV UL13 inhibits cGAS–STING-mediated IFN-β production by phosphorylating IRF3

Exploits des François

Aryl hydrocarbon receptor suppresses STING-mediated type I IFN expression in triple-negative breast cancer

Monocyte-derived macrophages aggravate pulmonary vasculitis via cGAS/STING/IFN-mediated nucleic acid sensing

SGLT2 inhibitor activates the STING/IRF3/IFN-β pathway and induces immune infiltration in osteosarcoma

Sting

Sting

STING induces early IFN-beta in the liver and constrains myeloid cell-mediated dissemination of murine cytomegalovirus

Influenza C virus NS1 protein counteracts RIG-I-mediated IFN signalling

DUSP4 modulates RIG-I- and STING-mediated IRF3-type I IFN response

TLR9 and STING agonists synergistically induce innate and adaptive type‐II IFN

ARF1 controls STING activation and recycling to prevent chronic type I IFN signalling

PRV UL13 inhibits cGAS–STING-mediated IFN-β production by phosphorylating IRF3

Exploits des François

Aryl hydrocarbon receptor suppresses STING-mediated type I IFN expression in triple-negative breast cancer

Monocyte-derived macrophages aggravate pulmonary vasculitis via cGAS/STING/IFN-mediated nucleic acid sensing

SGLT2 inhibitor activates the STING/IRF3/IFN-β pathway and induces immune infiltration in osteosarcoma

Sting

Sting

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