Targeting Fn14 as a therapeutic target for cachexia reprograms the glycolytic pathway in tumour and brain in mice
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
-
Targeting Fn14 as a therapeutic target for cachexia reprograms the glycolytic pathway in tumour and brain in mice ; day:26 ; month:7 ; year:2024 ; pages:1-9
European journal of nuclear medicine and molecular imaging ; (26.7.2024), 1-9
- Creator
-
Burvenich, Ingrid Julienne Georgette
Osellame, Laura Danielle
Rigopoulos, Angela
Huynh, Nhi
Cao, Zhipeng
Hoogenraad, Nicholas Johannes
Scott, Andrew Mark
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1007/s00259-024-06836-1
- URN
-
urn:nbn:de:101:1-2410112115365.279819673812
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:29 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Burvenich, Ingrid Julienne Georgette
- Osellame, Laura Danielle
- Rigopoulos, Angela
- Huynh, Nhi
- Cao, Zhipeng
- Hoogenraad, Nicholas Johannes
- Scott, Andrew Mark
- SpringerLink (Online service)
Other Objects (12)
Bedaquiline reprograms central metabolism to reveal glycolytic vulnerability in Mycobacterium tuberculosis
Vascular promotion reprograms the tumour microenvironment toward enhanced drug delivery
Trans-vaccenic acid reprograms CD8+ T cells and anti-tumour immunity
Activation of GPR81 by lactate drives tumour-induced cachexia
Tumour-originated exosomal miR-155 triggers cancer-associated cachexia to promote tumour progression
Challenges in Targeting the “Crosstalks” in Cancer Cachexia
Retraction Note: Tumour-originated exosomal miR-155 triggers cancer-associated cachexia to promote tumour progression
Cachexia
Cachexia
Targeting WDxR motif reprograms immune microenvironment and inhibits hepatocellular carcinoma progression
The potential of hyperpolarised 13C-MRI to target glycolytic tumour core in prostate cancer
Enhanced tumour cell nuclear targeting in a tumour progression model
Bedaquiline reprograms central metabolism to reveal glycolytic vulnerability in Mycobacterium tuberculosis
Vascular promotion reprograms the tumour microenvironment toward enhanced drug delivery
Trans-vaccenic acid reprograms CD8+ T cells and anti-tumour immunity
Activation of GPR81 by lactate drives tumour-induced cachexia
Tumour-originated exosomal miR-155 triggers cancer-associated cachexia to promote tumour progression
Challenges in Targeting the “Crosstalks” in Cancer Cachexia
Retraction Note: Tumour-originated exosomal miR-155 triggers cancer-associated cachexia to promote tumour progression
Cachexia
Cachexia
Targeting WDxR motif reprograms immune microenvironment and inhibits hepatocellular carcinoma progression
The potential of hyperpolarised 13C-MRI to target glycolytic tumour core in prostate cancer
Enhanced tumour cell nuclear targeting in a tumour progression model
Bedaquiline reprograms central metabolism to reveal glycolytic vulnerability in Mycobacterium tuberculosis
Vascular promotion reprograms the tumour microenvironment toward enhanced drug delivery
Trans-vaccenic acid reprograms CD8+ T cells and anti-tumour immunity
Activation of GPR81 by lactate drives tumour-induced cachexia
Tumour-originated exosomal miR-155 triggers cancer-associated cachexia to promote tumour progression
Challenges in Targeting the “Crosstalks” in Cancer Cachexia
Retraction Note: Tumour-originated exosomal miR-155 triggers cancer-associated cachexia to promote tumour progression
Cachexia
Cachexia
Targeting WDxR motif reprograms immune microenvironment and inhibits hepatocellular carcinoma progression
The potential of hyperpolarised 13C-MRI to target glycolytic tumour core in prostate cancer