Blocking PDGF-CC signaling ameliorates multiple sclerosis-like neuroinflammation by inhibiting disruption of the blood–brain barrier

to related objects

Location
Deutsche Nationalbibliothek Frankfurt am Main
ISSN
2045-2322
Extent
Online-Ressource
Language
Englisch
Notes
online resource.

Bibliographic citation
Blocking PDGF-CC signaling ameliorates multiple sclerosis-like neuroinflammation by inhibiting disruption of the blood–brain barrier ; volume:10 ; number:1 ; day:24 ; month:12 ; year:2020 ; pages:1-21 ; date:12.2020
Scientific reports ; 10, Heft 1 (24.12.2020), 1-21, 12.2020

Creator
Zeitelhofer, Manuel
Adzemovic, Milena Z.
Moessinger, Christine
Stefanitsch, Christina
Strell, Carina
Muhl, Lars
Brundin, Lou
Fredriksson, Linda
Olsson, Tomas
Eriksson, Ulf
Nilsson, Ingrid
Contributor
SpringerLink (Online service)

DOI
10.1038/s41598-020-79598-z
URN
urn:nbn:de:101:1-2021012420124225658698
Rights
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
Last update
15.08.2025, 7:37 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Show original at data provider

Associated

  • Zeitelhofer, Manuel
  • Adzemovic, Milena Z.
  • Moessinger, Christine
  • Stefanitsch, Christina
  • Strell, Carina
  • Muhl, Lars
  • Brundin, Lou
  • Fredriksson, Linda
  • Olsson, Tomas
  • Eriksson, Ulf
  • Nilsson, Ingrid
  • SpringerLink (Online service)

Other Objects (12)

Epigenetics in blood–brain barrier disruption

Epigenetics in blood–brain barrier disruption

Consequences of blood-brain barrier disruption

Hochschulschrift

Consequences of blood-brain barrier disruption

Adjudin protects against cerebral ischemia reperfusion injury by inhibition of neuroinflammation and blood-brain barrier disruption

Adjudin protects against cerebral ischemia reperfusion injury by inhibition of neuroinflammation and blood-brain barrier disruption

Meningitic Escherichia coli-induced upregulation of PDGF-B and ICAM-1 aggravates blood-brain barrier disruption and neuroinflammatory response

Meningitic Escherichia coli-induced upregulation of PDGF-B and ICAM-1 aggravates blood-brain barrier disruption and neuroinflammatory response

Carbon monoxide-releasing molecule-3 protects against ischemic stroke by suppressing neuroinflammation and alleviating blood-brain barrier disruption

Carbon monoxide-releasing molecule-3 protects against ischemic stroke by suppressing neuroinflammation and alleviating blood-brain barrier disruption

Compromised endothelial Wnt/β-catenin signaling mediates the blood-brain barrier disruption and leads to neuroinflammation in endotoxemia

Compromised endothelial Wnt/β-catenin signaling mediates the blood-brain barrier disruption and leads to neuroinflammation in endotoxemia

Neuro-inflammation, blood-brain barrier, seizures and autism

Neuro-inflammation, blood-brain barrier, seizures and autism

Blood-brain barrier disruption: a culprit of cognitive decline?

Blood-brain barrier disruption: a culprit of cognitive decline?

Breaking barriers: noncanonical inflammasome executes blood–brain barrier disruption

Breaking barriers: noncanonical inflammasome executes blood–brain barrier disruption

Cavitation dose painting for focused ultrasound-induced blood-brain barrier disruption

Cavitation dose painting for focused ultrasound-induced blood-brain barrier disruption

Neurovascular unit disruption and blood–brain barrier leakage in MCT8 deficiency

Neurovascular unit disruption and blood–brain barrier leakage in MCT8 deficiency

JNK signaling is the shared pathway linking neuroinflammation, blood–brain barrier disruption, and oligodendroglial apoptosis in the white matter injury of the immature brain

JNK signaling is the shared pathway linking neuroinflammation, blood–brain barrier disruption, and oligodendroglial apoptosis in the white matter injury of the immature brain

Related objects