miR-451a abrogates treatment resistance in FLT3-ITD-positive acute myeloid leukemia
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2044-5385
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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miR-451a abrogates treatment resistance in FLT3-ITD-positive acute myeloid leukemia ; volume:8 ; number:3 ; day:21 ; month:3 ; year:2018 ; pages:1-4 ; date:3.2018
Blood cancer journal ; 8, Heft 3 (21.3.2018), 1-4, 3.2018
- Creator
- Contributor
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Wurm, Alexander A.
Gerloff, Dennis
Katzerke, Christiane
Bräuer-Hartmann, Daniela
Hartmann, Jens-Uwe
Wilke, Franziska
Thiede, Christian
Müller-Tidow, Carsten
Niederwieser, Dietger
Behre, Gerhard
SpringerLink (Online service)
- DOI
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10.1038/s41408-018-0070-y
- URN
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urn:nbn:de:101:1-2018053000370888497225
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:23 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Krakowsky, Rosanna Henriette Elise
- Wurm, Alexander A.
- Gerloff, Dennis
- Katzerke, Christiane
- Bräuer-Hartmann, Daniela
- Hartmann, Jens-Uwe
- Wilke, Franziska
- Thiede, Christian
- Müller-Tidow, Carsten
- Niederwieser, Dietger
- Behre, Gerhard
- SpringerLink (Online service)
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Receptor protein-tyrosine phosphatases controlling activity of the oncoprotein FLT3 ITD
Identification of RPS6KA1 as a mediator of resistance to Venetoclax/Azacytidine-combination treatment in Acute Myeloid Leukemia by genome-scale CRISPR/Cas9 knockout screening
Expression of protein-tyrosine phosphatases in Acute Myeloid Leukemia cells: FLT3 ITD sustains high levels of DUSP6 expression
RSK1 dependency in FLT3-ITD acute myeloid leukemia
The landscape of small nucleolar RNA expression and potential roles of RNA modifications in multiple myeloma
Expressionsanalyse und funktionelle Charakterisierung von Zyklin A1 und der Interaktionspartner des Zyklin A1-CDK2-Komplexes
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Autophagy activation mediates resistance to FLT3 inhibitors in acute myeloid leukemia with FLT3-ITD mutation
The role of GAB2 in FLT3-ITD mutant acute myeloid leukemia
Gab2 deficiency prevents Flt3-ITD driven acute myeloid leukemia in vivo
A dual inhibitor overcomes drug-resistant FLT3-ITD acute myeloid leukemia
The cooperative effect of IL3RB and FLT3-ITD in acute myeloid leukemia
Receptor protein-tyrosine phosphatases controlling activity of the oncoprotein FLT3 ITD
Identification of RPS6KA1 as a mediator of resistance to Venetoclax/Azacytidine-combination treatment in Acute Myeloid Leukemia by genome-scale CRISPR/Cas9 knockout screening
Expression of protein-tyrosine phosphatases in Acute Myeloid Leukemia cells: FLT3 ITD sustains high levels of DUSP6 expression
RSK1 dependency in FLT3-ITD acute myeloid leukemia
The landscape of small nucleolar RNA expression and potential roles of RNA modifications in multiple myeloma
Expressionsanalyse und funktionelle Charakterisierung von Zyklin A1 und der Interaktionspartner des Zyklin A1-CDK2-Komplexes
Distinct Metabolic features differentiating FLT3-ITD AML from FLT3-WT childhood Acute Myeloid Leukemia
Autophagy activation mediates resistance to FLT3 inhibitors in acute myeloid leukemia with FLT3-ITD mutation
The role of GAB2 in FLT3-ITD mutant acute myeloid leukemia
Gab2 deficiency prevents Flt3-ITD driven acute myeloid leukemia in vivo
A dual inhibitor overcomes drug-resistant FLT3-ITD acute myeloid leukemia