Convergent evolution and multi-wave clonal invasion in H3 K27-altered diffuse midline gliomas treated with a PDGFR inhibitor
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2051-5960
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Convergent evolution and multi-wave clonal invasion in H3 K27-altered diffuse midline gliomas treated with a PDGFR inhibitor ; volume:10 ; number:1 ; day:31 ; month:5 ; year:2022 ; pages:1-14 ; date:12.2022
Acta Neuropathologica Communications ; 10, Heft 1 (31.5.2022), 1-14, 12.2022
- Urheber
-
Arunachalam, Sasi
Szlachta, Karol
Brady, Samuel W.
Ma, Xiaotu
Ju, Bensheng
Shaner, Bridget
Mulder, Heather L.
Easton, John
Raphael, Benjamin J.
Myers, Matthew
Tinkle, Christopher
Allen, Sariah J.
Orr, Brent A.
Wetmore, Cynthia J.
Baker, Suzanne J.
Zhang, Jinghui
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/s40478-022-01381-0
- URN
-
urn:nbn:de:101:1-2022073107114250785797
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:24 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Arunachalam, Sasi
- Szlachta, Karol
- Brady, Samuel W.
- Ma, Xiaotu
- Ju, Bensheng
- Shaner, Bridget
- Mulder, Heather L.
- Easton, John
- Raphael, Benjamin J.
- Myers, Matthew
- Tinkle, Christopher
- Allen, Sariah J.
- Orr, Brent A.
- Wetmore, Cynthia J.
- Baker, Suzanne J.
- Zhang, Jinghui
- SpringerLink (Online service)
Ähnliche Objekte (12)
INTERCEPT H3: a multicenter phase I peptide vaccine trial for the treatment of H3-mutated diffuse midline gliomas
Incidence and clinicopathologic features of H3 K27M mutations in adults with radiographically-determined midline gliomas
Infiltrative gliomas of the thalamus in children: the role of surgery in the era of H3 K27M mutant midline gliomas
H3K27M-Altered Diffuse Midline Gliomas: A Review
MRI comparative study of diffuse midline glioma, H3 K27-altered and glioma in the midline without H3 K27-altered
Integrated analyses reveal two molecularly and clinically distinct subtypes of H3 K27M-mutant diffuse midline gliomas with prognostic significance
EZHIP is a specific diagnostic biomarker for posterior fossa ependymomas, group PFA and diffuse midline gliomas H3-WT with EZHIP overexpression
Diffuse Mittelliniengliome, H3 K27M-mutiert sind im Vergleich zu Glioblastomen, H3 K27M-Wildtyp von Mittellinienstrukturen mit einem geringeren perifokalen Ödem und Kontrastmittelaufnahme assoziiert : = Diffuse midline gliomas, H3 K27M-mutant are associated with less peritumoral edema and contrast enhancement in comparison to glioblastomas, H3 K27M wildtype of midline structures
Transcriptomic and epigenetic profiling of ‘diffuse midline gliomas, H3 K27M-mutant’ discriminate two subgroups based on the type of histone H3 mutated and not supratentorial or infratentorial location
Absence of MGMT promoter methylation in diffuse midline glioma, H3 K27M-mutant
Imaging features associated with H3 K27-altered and H3 G34-mutant gliomas: a narrative systematic review
Defining tumor-associated vascular heterogeneity in pediatric high-grade and diffuse midline gliomas
INTERCEPT H3: a multicenter phase I peptide vaccine trial for the treatment of H3-mutated diffuse midline gliomas
Incidence and clinicopathologic features of H3 K27M mutations in adults with radiographically-determined midline gliomas
Infiltrative gliomas of the thalamus in children: the role of surgery in the era of H3 K27M mutant midline gliomas
H3K27M-Altered Diffuse Midline Gliomas: A Review
MRI comparative study of diffuse midline glioma, H3 K27-altered and glioma in the midline without H3 K27-altered
Integrated analyses reveal two molecularly and clinically distinct subtypes of H3 K27M-mutant diffuse midline gliomas with prognostic significance
EZHIP is a specific diagnostic biomarker for posterior fossa ependymomas, group PFA and diffuse midline gliomas H3-WT with EZHIP overexpression
Diffuse Mittelliniengliome, H3 K27M-mutiert sind im Vergleich zu Glioblastomen, H3 K27M-Wildtyp von Mittellinienstrukturen mit einem geringeren perifokalen Ödem und Kontrastmittelaufnahme assoziiert : = Diffuse midline gliomas, H3 K27M-mutant are associated with less peritumoral edema and contrast enhancement in comparison to glioblastomas, H3 K27M wildtype of midline structures
Transcriptomic and epigenetic profiling of ‘diffuse midline gliomas, H3 K27M-mutant’ discriminate two subgroups based on the type of histone H3 mutated and not supratentorial or infratentorial location
Absence of MGMT promoter methylation in diffuse midline glioma, H3 K27M-mutant
Imaging features associated with H3 K27-altered and H3 G34-mutant gliomas: a narrative systematic review
Defining tumor-associated vascular heterogeneity in pediatric high-grade and diffuse midline gliomas
INTERCEPT H3: a multicenter phase I peptide vaccine trial for the treatment of H3-mutated diffuse midline gliomas
Incidence and clinicopathologic features of H3 K27M mutations in adults with radiographically-determined midline gliomas
Infiltrative gliomas of the thalamus in children: the role of surgery in the era of H3 K27M mutant midline gliomas
H3K27M-Altered Diffuse Midline Gliomas: A Review
MRI comparative study of diffuse midline glioma, H3 K27-altered and glioma in the midline without H3 K27-altered
Integrated analyses reveal two molecularly and clinically distinct subtypes of H3 K27M-mutant diffuse midline gliomas with prognostic significance
EZHIP is a specific diagnostic biomarker for posterior fossa ependymomas, group PFA and diffuse midline gliomas H3-WT with EZHIP overexpression
Diffuse Mittelliniengliome, H3 K27M-mutiert sind im Vergleich zu Glioblastomen, H3 K27M-Wildtyp von Mittellinienstrukturen mit einem geringeren perifokalen Ödem und Kontrastmittelaufnahme assoziiert : = Diffuse midline gliomas, H3 K27M-mutant are associated with less peritumoral edema and contrast enhancement in comparison to glioblastomas, H3 K27M wildtype of midline structures
Transcriptomic and epigenetic profiling of ‘diffuse midline gliomas, H3 K27M-mutant’ discriminate two subgroups based on the type of histone H3 mutated and not supratentorial or infratentorial location
Absence of MGMT promoter methylation in diffuse midline glioma, H3 K27M-mutant
Imaging features associated with H3 K27-altered and H3 G34-mutant gliomas: a narrative systematic review