Mitochondrial Ca2+ uptake in skeletal muscle health and disease

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1869-1889

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Mitochondrial Ca2+ uptake in skeletal muscle health and disease ; day:19 ; month:7 ; year:2016 ; pages:1-7
Science China / Life sciences. Life sciences ; (19.7.2016), 1-7

Creator: Zhou, Jingsong

Contributor: Dhakal, Kamal
Yi, Jianxun
SpringerLink (Online service)

DOI: 10.1007/s11427-016-5089-3

URN: urn:nbn:de:1111-2016072215127

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:45 AM CEST

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Zhou, Jingsong
Dhakal, Kamal
Yi, Jianxun
SpringerLink (Online service)

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Mitochondrial Ca2+ uptake in skeletal muscle health and disease

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Other Objects (12)

Mitochondrial dysfunction: roles in skeletal muscle atrophy

Absence of physiological Ca2+ transients is an initial trigger for mitochondrial dysfunction in skeletal muscle following denervation

Ca2+ permeation and/or binding to CaV1.1 fine-tunes skeletal muscle Ca2+ signaling to sustain muscle function

Mitochondrial glycerol 3‐phosphate dehydrogenase promotes skeletal muscle regeneration

Alteration in mitochondrial Ca2+ uptake disrupts insulin signaling in hypertrophic cardiomyocytes

Skeletal muscle

Skeletal muscle

Altered skeletal muscle (mitochondrial) properties in patients with mitochondrial DNA single deletion myopathy

Ca2+ and force during dynamic contractions in mouse intact skeletal muscle fibers

Exercise-Regulated Mitochondrial and Nuclear Signalling Networks in Skeletal Muscle

Longitudinal associations between blood lysophosphatidylcholines and skeletal muscle mitochondrial function

Insulin stimulates β-alanine uptake in skeletal muscle cells in vitro

Mitochondrial dysfunction: roles in skeletal muscle atrophy

Absence of physiological Ca2+ transients is an initial trigger for mitochondrial dysfunction in skeletal muscle following denervation

Ca2+ permeation and/or binding to CaV1.1 fine-tunes skeletal muscle Ca2+ signaling to sustain muscle function

Mitochondrial glycerol 3‐phosphate dehydrogenase promotes skeletal muscle regeneration

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Skeletal muscle

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Longitudinal associations between blood lysophosphatidylcholines and skeletal muscle mitochondrial function

Insulin stimulates β-alanine uptake in skeletal muscle cells in vitro

Mitochondrial dysfunction: roles in skeletal muscle atrophy

Absence of physiological Ca2+ transients is an initial trigger for mitochondrial dysfunction in skeletal muscle following denervation

Ca2+ permeation and/or binding to CaV1.1 fine-tunes skeletal muscle Ca2+ signaling to sustain muscle function

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Alteration in mitochondrial Ca2+ uptake disrupts insulin signaling in hypertrophic cardiomyocytes

Skeletal muscle

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Altered skeletal muscle (mitochondrial) properties in patients with mitochondrial DNA single deletion myopathy

Ca2+ and force during dynamic contractions in mouse intact skeletal muscle fibers

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Longitudinal associations between blood lysophosphatidylcholines and skeletal muscle mitochondrial function

Insulin stimulates β-alanine uptake in skeletal muscle cells in vitro

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