A novel splicing variant of ANXA11 in a patient with amyotrophic lateral sclerosis: histologic and biochemical features
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2051-5960
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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A novel splicing variant of ANXA11 in a patient with amyotrophic lateral sclerosis: histologic and biochemical features ; volume:9 ; number:1 ; day:7 ; month:6 ; year:2021 ; pages:1-9 ; date:12.2021
Acta Neuropathologica Communications ; 9, Heft 1 (7.6.2021), 1-9, 12.2021
- Creator
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Sainouchi, Makoto
Hatano, Yuya
Tada, Mari
Ishihara, Tomohiko
Ando, Shoichiro
Kato, Taisuke
Tokunaga, Jun
Ito, Gaku
Miyahara, Hiroaki
Toyoshima, Yasuko
Yokoseki, Akio
Ozawa, Tetsutaro
Akazawa, Kohei
Onodera, Osamu
Kakita, Akiyoshi
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s40478-021-01202-w
- URN
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urn:nbn:de:101:1-2021101020231317495865
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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2025-08-15T07:37:41+0200
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Sainouchi, Makoto
- Hatano, Yuya
- Tada, Mari
- Ishihara, Tomohiko
- Ando, Shoichiro
- Kato, Taisuke
- Tokunaga, Jun
- Ito, Gaku
- Miyahara, Hiroaki
- Toyoshima, Yasuko
- Yokoseki, Akio
- Ozawa, Tetsutaro
- Akazawa, Kohei
- Onodera, Osamu
- Kakita, Akiyoshi
- SpringerLink (Online service)
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Correction to: A novel splicing variant of ANXA11 in a patient with amyotrophic lateral sclerosis: histologic and biochemical features
CD147 expression predicts biochemical recurrence after prostatectomy independent of histologic and pathologic features
The ATRX splicing variant c.21-1G>A is asymptomatic
Aberrant alternative splicing: the molecular hallmark of sporadic amyotrophic lateral sclerosis and beyond
Aberrant NOVA1 function disrupts alternative splicing in early stages of amyotrophic lateral sclerosis
Benchmarking splice variant prediction algorithms using massively parallel splicing assays
Functional analysis of human hyaluronan synthase 3 splicing variant 2
Identification of a STIM1 Splicing Variant that Promotes Glioblastoma Growth
Biochemical characterization of a disease-causing human osteoprotegerin variant
A silent COL17A1 variant alters splicing and causes junctional epidermolysis bullosa
Slowly progressive autosomal dominant Alport Syndrome due to COL4A3 splicing variant
Supervised Regularized Canonical Correlation Analysis: integrating histologic and proteomic measurements for predicting biochemical recurrence following prostate surgery
Correction to: A novel splicing variant of ANXA11 in a patient with amyotrophic lateral sclerosis: histologic and biochemical features
CD147 expression predicts biochemical recurrence after prostatectomy independent of histologic and pathologic features
The ATRX splicing variant c.21-1G>A is asymptomatic
Aberrant alternative splicing: the molecular hallmark of sporadic amyotrophic lateral sclerosis and beyond
Aberrant NOVA1 function disrupts alternative splicing in early stages of amyotrophic lateral sclerosis
Benchmarking splice variant prediction algorithms using massively parallel splicing assays
Functional analysis of human hyaluronan synthase 3 splicing variant 2
Identification of a STIM1 Splicing Variant that Promotes Glioblastoma Growth
Biochemical characterization of a disease-causing human osteoprotegerin variant
A silent COL17A1 variant alters splicing and causes junctional epidermolysis bullosa
Slowly progressive autosomal dominant Alport Syndrome due to COL4A3 splicing variant