The role of Xist ‐mediated Polycomb recruitment in the initiation of X‐chromosome inactivation
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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The role of Xist ‐mediated Polycomb recruitment in the initiation of X‐chromosome inactivation ; volume:20 ; number:10 ; year:2019 ; extent:18
EMBO reports / European Molecular Biology Organization ; 20, Heft 10 (2019) (gesamt 18)
- Creator
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Bousard, Aurélie
Raposo, Ana Cláudia
Żylicz, Jan Jakub
Picard, Christel
Pires, Vanessa Borges
Qi, Yanyan
Gil, Cláudia
Syx, Laurène
Chang, Howard Y.
Heard, Edith
da Rocha, Simão Teixeira
- DOI
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10.15252/embr.201948019
- URN
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urn:nbn:de:101:1-2022072508484126538806
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:28 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Bousard, Aurélie
- Raposo, Ana Cláudia
- Żylicz, Jan Jakub
- Picard, Christel
- Pires, Vanessa Borges
- Qi, Yanyan
- Gil, Cláudia
- Syx, Laurène
- Chang, Howard Y.
- Heard, Edith
- da Rocha, Simão Teixeira
Other Objects (12)
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The complex story of human X chromosome inactivation
Random X chromosome inactivation in patients with Klinefelter syndrome
Skewed X-chromosome inactivation in patients with esophageal carcinoma
Incomplete meiotic sex chromosome inactivation in the domestic dog
Methylation status of genes escaping from X-chromosome inactivation in patients with X-chromosome rearrangements
Attrition of X chromosome inactivation in aged hematopoietic stem cells
Population variability in X-chromosome inactivation across 10 mammalian species
SUMOylated non-canonical polycomb PRC1.6 complex as a prerequisite for recruitment of transcription factor RBPJ
Sustained inactivation of the Polycomb PRC1 complex induces DNA repair defects and genomic instability in epigenetic tumors
Imprinted X chromosome inactivation: evolution of mechanisms in distantly related mammals
A system to analyze the initiation of random X-chromosome inactivation using time-lapse imaging of single cells
Transcriptome regulation during the X chromosome inactivation process
The complex story of human X chromosome inactivation
Random X chromosome inactivation in patients with Klinefelter syndrome
Skewed X-chromosome inactivation in patients with esophageal carcinoma
Incomplete meiotic sex chromosome inactivation in the domestic dog
Methylation status of genes escaping from X-chromosome inactivation in patients with X-chromosome rearrangements
Attrition of X chromosome inactivation in aged hematopoietic stem cells
Population variability in X-chromosome inactivation across 10 mammalian species
SUMOylated non-canonical polycomb PRC1.6 complex as a prerequisite for recruitment of transcription factor RBPJ
Sustained inactivation of the Polycomb PRC1 complex induces DNA repair defects and genomic instability in epigenetic tumors
Imprinted X chromosome inactivation: evolution of mechanisms in distantly related mammals
A system to analyze the initiation of random X-chromosome inactivation using time-lapse imaging of single cells
Transcriptome regulation during the X chromosome inactivation process
The complex story of human X chromosome inactivation
Random X chromosome inactivation in patients with Klinefelter syndrome
Skewed X-chromosome inactivation in patients with esophageal carcinoma
Incomplete meiotic sex chromosome inactivation in the domestic dog
Methylation status of genes escaping from X-chromosome inactivation in patients with X-chromosome rearrangements
Attrition of X chromosome inactivation in aged hematopoietic stem cells
Population variability in X-chromosome inactivation across 10 mammalian species
SUMOylated non-canonical polycomb PRC1.6 complex as a prerequisite for recruitment of transcription factor RBPJ
Sustained inactivation of the Polycomb PRC1 complex induces DNA repair defects and genomic instability in epigenetic tumors