CRISPR/Cas9 genome editing of CCR5 combined with C46 HIV-1 fusion inhibitor for cellular resistant to R5 and X4 tropic HIV-1
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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CRISPR/Cas9 genome editing of CCR5 combined with C46 HIV-1 fusion inhibitor for cellular resistant to R5 and X4 tropic HIV-1 ; volume:14 ; number:1 ; day:13 ; month:5 ; year:2024 ; pages:1-13 ; date:12.2024
Scientific reports ; 14, Heft 1 (13.5.2024), 1-13, 12.2024
- Creator
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Khamaikawin, Wannisa
Saisawang, Chonticha
Tassaneetrithep, Boonrat
Bhukhai, Kanit
Phanthong, Phetcharat
Borwornpinyo, Suparerk
Phuphuakrat, Angsana
Pasomsub, Ekawat
Chaisavaneeyakorn, Sujittra
Anurathapan, Usanarat
Apiwattanakul, Nopporn
Hongeng, Suradej
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41598-024-61626-x
- URN
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urn:nbn:de:101:1-2407222059536.415549526564
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:57 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Khamaikawin, Wannisa
- Saisawang, Chonticha
- Tassaneetrithep, Boonrat
- Bhukhai, Kanit
- Phanthong, Phetcharat
- Borwornpinyo, Suparerk
- Phuphuakrat, Angsana
- Pasomsub, Ekawat
- Chaisavaneeyakorn, Sujittra
- Anurathapan, Usanarat
- Apiwattanakul, Nopporn
- Hongeng, Suradej
- SpringerLink (Online service)
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Activity and structural analysis of GRL-117C: a novel small molecule CCR5 inhibitor active against R5-tropic HIV-1s
Spermine and spermidine bind CXCR4 and inhibit CXCR4- but not CCR5-tropic HIV-1 infection
Neutralization of X4- and R5-tropic HIV-1 NL4-3 variants by HOCl-modified serum albumins
Infection of ectocervical tissue and universal targeting of T-cells mediated by primary non-macrophage-tropic and highly macrophage-tropic HIV-1 R5 envelopes
Vpx complementation of ‘non-macrophage tropic’ R5 viruses reveals robust entry of infectious HIV-1 cores into macrophages
HIV-1 non-macrophage-tropic R5 envelope glycoproteins are not more tropic for entry into primary CD4+ T-cells than envelopes highly adapted for macrophages
Development of a CCR5-tropic HIV-1 fusion inhibition assay amenable to high-throughput screening for topical microbicides
RT-SHIV, an infectious CCR5-tropic chimeric virus suitable for evaluating HIV reverse transcriptase inhibitors in macaque models
Prevalence of CCR5-tropic HIV-1 among treatment-experienced individuals in South Africa infected with Clade C virus
Genome editing of the HIV co-receptors CCR5 and CXCR4 by CRISPR-Cas9 protects CD4+ T cells from HIV-1 infection
Reduced CCR5 expression among Uganda HIV controllers
Monophylogenetic HIV-1C epidemic in Ethiopia is dominated by CCR5-tropic viruses–an analysis of a prospective country-wide cohort
Activity and structural analysis of GRL-117C: a novel small molecule CCR5 inhibitor active against R5-tropic HIV-1s
Spermine and spermidine bind CXCR4 and inhibit CXCR4- but not CCR5-tropic HIV-1 infection
Neutralization of X4- and R5-tropic HIV-1 NL4-3 variants by HOCl-modified serum albumins
Infection of ectocervical tissue and universal targeting of T-cells mediated by primary non-macrophage-tropic and highly macrophage-tropic HIV-1 R5 envelopes
Vpx complementation of ‘non-macrophage tropic’ R5 viruses reveals robust entry of infectious HIV-1 cores into macrophages
HIV-1 non-macrophage-tropic R5 envelope glycoproteins are not more tropic for entry into primary CD4+ T-cells than envelopes highly adapted for macrophages
Development of a CCR5-tropic HIV-1 fusion inhibition assay amenable to high-throughput screening for topical microbicides
RT-SHIV, an infectious CCR5-tropic chimeric virus suitable for evaluating HIV reverse transcriptase inhibitors in macaque models
Prevalence of CCR5-tropic HIV-1 among treatment-experienced individuals in South Africa infected with Clade C virus
Genome editing of the HIV co-receptors CCR5 and CXCR4 by CRISPR-Cas9 protects CD4+ T cells from HIV-1 infection
Reduced CCR5 expression among Uganda HIV controllers