Correction: Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Correction: Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells ; volume:11 ; number:1 ; day:26 ; month:10 ; year:2023 ; pages:1 ; date:12.2023
Cancer & metabolism ; 11, Heft 1 (26.10.2023), 1, 12.2023
- Creator
-
Mayengbam, Shyamananda Singh
Singh, Abhijeet
Yaduvanshi, Himanshi
Bhati, Firoz Khan
Deshmukh, Bhavana
Athavale, Dipti
Ramteke, Pranay L.
Bhat, Manoj Kumar
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s40170-023-00321-3
- URN
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urn:nbn:de:101:1-2024011511445339376645
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:34 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Mayengbam, Shyamananda Singh
- Singh, Abhijeet
- Yaduvanshi, Himanshi
- Bhati, Firoz Khan
- Deshmukh, Bhavana
- Athavale, Dipti
- Ramteke, Pranay L.
- Bhat, Manoj Kumar
- SpringerLink (Online service)
Other Objects (12)
Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells
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Med23 deficiency reprograms the tumor microenvironment to promote lung tumorigenesis
Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis
Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis
IGF2BP2-meidated m6A modification of CSF2 reprograms MSC to promote gastric cancer progression
PIM1 drives lipid droplet accumulation to promote proliferation and survival in prostate cancer
Progression of prostate cancer reprograms MYC-mediated lipid metabolism via lysine methyltransferase 2A
Overexpression of lncRNA IGFBP4–1 reprograms energy metabolism to promote lung cancer progression
UBR5 metabolically reprograms nasopharyngeal carcinoma cells to promote glycolysis and M2 polarization via SPLUNC1 signaling
PILRB potentiates the PI3K/AKT signaling pathway and reprograms cholesterol metabolism to drive gastric tumorigenesis and metastasis
Mitochondrial oxidative damage reprograms lipid metabolism of renal tubular epithelial cells in the diabetic kidney
Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells
Fatty-acid-induced FABP5/HIF-1 reprograms lipid metabolism and enhances the proliferation of liver cancer cells
Med23 deficiency reprograms the tumor microenvironment to promote lung tumorigenesis
Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis
Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis
IGF2BP2-meidated m6A modification of CSF2 reprograms MSC to promote gastric cancer progression
PIM1 drives lipid droplet accumulation to promote proliferation and survival in prostate cancer
Progression of prostate cancer reprograms MYC-mediated lipid metabolism via lysine methyltransferase 2A
Overexpression of lncRNA IGFBP4–1 reprograms energy metabolism to promote lung cancer progression
UBR5 metabolically reprograms nasopharyngeal carcinoma cells to promote glycolysis and M2 polarization via SPLUNC1 signaling
PILRB potentiates the PI3K/AKT signaling pathway and reprograms cholesterol metabolism to drive gastric tumorigenesis and metastasis
Mitochondrial oxidative damage reprograms lipid metabolism of renal tubular epithelial cells in the diabetic kidney
Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells
Fatty-acid-induced FABP5/HIF-1 reprograms lipid metabolism and enhances the proliferation of liver cancer cells
Med23 deficiency reprograms the tumor microenvironment to promote lung tumorigenesis
Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis
Author Correction: Efferocytosis reprograms the tumor microenvironment to promote pancreatic cancer liver metastasis
IGF2BP2-meidated m6A modification of CSF2 reprograms MSC to promote gastric cancer progression
PIM1 drives lipid droplet accumulation to promote proliferation and survival in prostate cancer
Progression of prostate cancer reprograms MYC-mediated lipid metabolism via lysine methyltransferase 2A
Overexpression of lncRNA IGFBP4–1 reprograms energy metabolism to promote lung cancer progression
UBR5 metabolically reprograms nasopharyngeal carcinoma cells to promote glycolysis and M2 polarization via SPLUNC1 signaling
PILRB potentiates the PI3K/AKT signaling pathway and reprograms cholesterol metabolism to drive gastric tumorigenesis and metastasis